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Inhibition of JNK and p38 MAPK-mediated inflammation and apoptosis by ivabradine improves cardiac function in streptozotocin-induced diabetic cardiomyopathy
Authors:Guangfeng Zuo  Xiaomin Ren  Xuesong Qian  Peng Ye  Jie Luo  Xiaofei Gao  Junjie Zhang  Shaoliang Chen
Affiliation:1. Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China

Zuo and Ren have contributed equally to this study.;2. Department of Cardiology, Zhangjiagang First People's Hospital, Zhangjiagang, China;3. Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China

Department of General Clinical Research Center, Nanjing First Hospital, Nanjing, China;4. Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China

Department of Basic Medicine, Key Laboratory of Cardiovascular Disease and Molecular Intervention, Nanjing Medical University, Nanjing, China;5. Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China

Abstract:
Keywords:diabetic cardiomyopathy (DCM)  inflammation  ivabradine
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