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Acetylcholinesterase and Nicotinic Acetylcholine Receptor Expression Diverge in Muscular Dysgenic Mice Lacking the L-Type Calcium Channel
Authors:Z. David Luo  Martine Pincon-Raymond   Palmer Taylor
Affiliation:Department of Pharmacology, University of California, San Diego, La Jolla, California, U.S.A.;and; CNRS 1283 Institut des Cordeliers, Paris, France
Abstract:
Keywords:Acetylcholinesterase expression    L-type Ca2+ channels    Muscular dysgenesis    mRNA stabilization    Nicotinic acetylcholine receptors    Muscle development
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