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Parental origin of germ-line and somatic mutations in the retinoblastoma gene
Authors:Kato  Mitsuo V  Ishizaki  Kanji  Shimizu  Takashi  Ejima  Yosuke  Tanooka  Hiroshi  Takayama  Jun  Kaneko  Akihiro  Toguchida  Junya  Sasaki  Masao S  Kato  M V
Institution:(1) Radiation Biology Center, Kyoto University, Yoshida-konoecho, Sakyo-ku, 606 Kyoto, Japan;(2) Laboratory of Molecular Oncology, Tsukuba Life Science Center, The Institute of Physical and Chemical Research (RIKEN), Koyadai, Tsukuba, 305 Ibaraki, Japan;(3) Radiobiology Division, National Cancer Center Research Institute, Tsukiji, Chuo-ku, 104 Tokyo, Japan;(4) Department of Pediatrics, National Cancer Center Hospital, Tsukiji, Chuo-ku, 104 Tokyo, Japan;(5) Department of Ophthalmology, National Cancer Center Hospital, Tsukiji, Chuo-ku, 104 Tokyo, Japan;(6) Department of Orthopaedic Surgery, Faculty of Medicine, Kyoto University, Shogoin-Kawaharacho, Sakyo-ku, 606 Kyoto, Japan
Abstract:Segregation analysis of polymorphic sites within the retinoblastoma (RB) gene and on chromosome 13, as well as the parental origin of the lost allele in the tumor, were analyzed in 24 families with RB patients. Four mutant alleles transmitted through the germ-line and seven de novo germ-line mutant alleles were identified in 11 patients with hereditary RB. Segregation analysis within the RB gene and on chromosome 13 was useful for DNA diagnosis of susceptibility to RB in relatives of hereditary patients, even if mutations were not identified. All seven de novo germ-line mutant alleles were paternally derived. The bias toward the paternal allele for de novo germ-line mutations of the RB gene was statistically significant. Seven paternal alleles and six maternal alleles were lost in 13 non-hereditary RB tumors with no bias in the parental origin of the somatic allele loss. These results suggest that the physical environment or a deficiency in DNA repair during spermatogenesis may be associated with significant risk factors for de novo germ-line mutations.
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