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SLC38A5 aggravates DC-mediated psoriasiform skin inflammation via potentiating lysosomal acidification
Affiliation:1. The Biomedical Translational Research Institute, Key Laboratory of Ministry of Education for Viral Pathogenesis & Infection Prevention and Control, Health Science Center (School of Medicine), Jinan University, Guangzhou 510632, China;2. Guangdong Provincial Key Laboratory of Tumor Interventional Diagnosis and Treatment, Zhuhai Institute of Translational Medicine, Zhuhai People’s Hospital Affiliated with Jinan University, Jinan University, Zhuhai 519000, China;3. Department of Dermatology, First Affiliated Hospital, Health Science Center (School of Medicine), Jinan University, Guangzhou 510632, China;4. Guangzhou National Laboratory, Guangzhou International BioIsland, Guangzhou 510005, China;5. Department of Dermatology, Guangdong Women’s and Children’s Hospital, Guangzhou 511442, China;6. Tsinghua-Berkeley Shenzhen Institute, Tsinghua University, Shenzhen 518055, China;7. The Sixth Affiliated Hospital of Guangzhou Medical University, Qingyuan People’s Hospital, Qingyuan 511518, China;8. Guangdong Provincial Key Laboratory of Virology, Institute of Medical Microbiology, Jinan University, Guangzhou 510632, China;9. Guandgong Provincial Key Laboratory of Spine and Spinal Cord Reconstruction, The Fifth Affiliated Hospital (Heyuan Shenhe People’s Hospital), Jinan University, Heyuan 517000, China;10. School of Biomedical Sciences, Li Ka Shing Faculty of Medicine, The University of Hong Kong, Hong Kong SAR, China
Abstract:
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  • Keywords:SLC38A5  psoriasis  lysosomal acidification  inflammation  dendritic cells  CP: Immunology
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