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Obesity of mice lacking VAP-1/SSAO by Aoc3 gene deletion is reproduced in mice expressing a mutated vascular adhesion protein-1 (VAP-1) devoid of amine oxidase activity
Authors:Jargaud  Valentin  Bour  Sandy  Tercé   François  Collet   Xavier  Valet   Philippe  Bouloumié   Anne  Guillemot   Jean-Claude  Mauriège  Pascale  Jalkanen  Sirpa  Stolen  Craig  Salmi  Marko  Smith  David J.  Carpéné   Christian
Affiliation:1.Institute of Metabolic and Cardiovascular Diseases, INSERM, UMR1048, Toulouse, France
;2.University of Toulouse, UMR1048, Paul Sabatier University, Toulouse, France
;3.Sanofi, Translational Sciences Unit, Chilly-Mazarin, France
;4.Dept. of Kinesiology, Fac. of Medicine and PEPS, Laval University, Québec, Canada
;5.MediCity and Institute of Biomedicine, University of Turku, Turku, Finland
;6.MediCity and Biotie Therapies Plc, Turku, Finland
;
Abstract:Journal of Physiology and Biochemistry - The product of Aoc3 gene is known as vascular adhesion protein-1 (VAP-1), a glycoprotein contributing to leukocyte extravasation and exhibiting...
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