Macrophage migration inhibitory factor enhances osteoclastogenesis through upregulation of RANKL expression from fibroblast-like synoviocytes in patients with rheumatoid arthritis期刊界 All Journals 搜尽天下杂志传播学术成果专业期刊搜索期刊信息化学术搜索

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Macrophage migration inhibitory factor enhances osteoclastogenesis through upregulation of RANKL expression from fibroblast-like synoviocytes in patients with rheumatoid arthritis

Authors:	Hae-Rim Kim Kyoung-Woon Kim Hong Geun Jung Kwang-Sup Yoon Hye-Jwa Oh Mi-La Cho Sang-Heon Lee

Institution:	(1) Division of Rheumatology, Medical Immunology Center, Department of Internal Medicine, Konkuk University School of Medicine, 1 Hwayang-dong, Kwangjin-gu, 143-729 Seoul, Korea;(2) Medical Immunology Center, Institute of Biomedical Science and Technology, Konkuk University, 1 Hwayang-dong, Kwangjin-gu, 143-729 Seoul, Korea;(3) Department of Orthopedic Surgery, Konkuk University School of Medicine, 1 Hwayang-dong, Kwangjin-gu, 143-729 Seoul, Korea;(4) The Rheumatism Research Center, Catholic Research Institute of Medical Science, The Catholic University of Korea, Seoul, South Korea;(5) The Rheumatism Research Center, Catholic Research Institute of Medical Science, The Catholic University of Korea, 505 Banpo-Dong, Seocho-Ku, 137-040 Seoul, Korea

Abstract:	Introduction Macrophage migration inhibitory factor (MIF) is one of key regulators in acute and chronic immune-inflammatory conditions including rheumatoid arthritis (RA). We examined the effect of MIF on osteoclastogenesis, which is known to play a crucial role in bone destruction in RA.

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