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The carboxyl terminus of the granulocyte colony-stimulating factor receptor, truncated in patients with severe congenital neutropenia/acute myeloid leukemia, is required for SH2-containing phosphatase-1 suppression of Stat activation.
Authors:F Dong  Y Qiu  T Yi  I P Touw  A C Larner
Institution:Department of Immunology, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, OH 44195, USA. dongf@ccf.org
Abstract:
Keywords:
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