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An IDH1-vitamin C crosstalk drives human erythroid development by inhibiting pro-oxidant mitochondrial metabolism
Authors:Pedro Gonzalez-Menendez  Manuela Romano  Hongxia Yan  Ruhi Deshmukh  Julien Papoin  Leal Oburoglu  Marie Daumur  Anne-Sophie Dumé  Ira Phadke  Cédric Mongellaz  Xiaoli Qu  Phuong-Nhi Bories  Michaela Fontenay  Xiuli An  Valérie Dardalhon  Marc Sitbon  Valérie S Zimmermann  Patrick G Gallagher  Sandrina Kinet
Institution:1. Institut de Génétique Moléculaire de Montpellier, Univ. Montpellier, CNRS, Montpellier, France;2. Laboratory of Excellence GR-Ex, Paris 75015, France;3. New York Blood Center, New York, NY, USA;4. Cancer Research UK Beatson Institute, Glasgow G61 1BD, UK;5. The Feinstein Institute for Medical Research, Manhasset, NY, USA;6. Pediatric Oncology Branch, NCI, CCR, NIH, Bethesda, MD, USA;7. Service d’Hématologie Biologique, Assistance Publique-Hôpitaux de Paris, Institut Cochin, Paris, France;8. Departments of Pediatrics and Genetics, Yale University School of Medicine, New Haven, CT, USA;9. Institute of Cancer Sciences, University of Glasgow, Glasgow G61 1QH, UK
Abstract:
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  • Keywords:erythropoiesis  hematopoietic stem and progenitor cell  vitamin C  isocitrate dehydrogenase  alpha-ketoglutarate  mitochondria  oxidative phosphorylation  redox stress  enucleation  human
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