The enhanced host-cell permissiveness of human cytomegalovirus is mediated by the Ras signaling pathway |
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Authors: | Harilaos FilippakisPanagiota Dimitropoulou Aristides G. EliopoulosDemetrios A. Spandidos George Sourvinos |
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Affiliation: | a Department of Clinical Virology, Faculty of Medicine, University of Crete, Greeceb Molecular and Cellular Laboratory, Faculty of Medicine, University of Crete, Greece |
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Abstract: | |
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Keywords: | DMEM, Dulbecco's Modified Eagle's Medium E, Early EBV, Epstein-Barr virus EGF, Epidermal Growth Factor EGFR, Epidermal Growth Factor Receptor ERK, Extracellular Regulated Kinase FBS, Fetal Bovine Serum FTI, Farnesyl Transferase Inhibitor HFF, Human Foreskin Fibroblasts HCMV, Human Cytomegalovirus HPI, Hours Post-Infection HHV, Human Herpes Virus HSV, Herpes Simplex Virus IE, Immediate Early IPTG, isopropyl-1thio-β-D galactoside L, Late MAPK, Mitogen Activated Protein Kinases MAPKK, MAPK kinase MAPKKK, MAPK kinase kinase MOI, Multiplicity of Infection NF-κB, Nuclear Factor kappa light chain enhancer of activated B cells PAA, Phosphono acetic Acid PFU, Plaque-Forming Units PI3-K, Phosphatidylinositol 3-Kinase PKR, RNA-activated protein kinase RALGDS, Ral Guanine Nucleotide Dissociation Stimulators SDS, Sodium-Dodecyl Sulphate TNF α, Tumor Necrosis Factor VZV, Varicella Zoster Virus |
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