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The enhanced host-cell permissiveness of human cytomegalovirus is mediated by the Ras signaling pathway
Authors:Harilaos FilippakisPanagiota Dimitropoulou  Aristides G. EliopoulosDemetrios A. Spandidos  George Sourvinos
Affiliation:
  • a Department of Clinical Virology, Faculty of Medicine, University of Crete, Greece
  • b Molecular and Cellular Laboratory, Faculty of Medicine, University of Crete, Greece
  • Abstract:
    Keywords:DMEM, Dulbecco's Modified Eagle's Medium   E, Early   EBV, Epstein-Barr virus   EGF, Epidermal Growth Factor   EGFR, Epidermal Growth Factor Receptor   ERK, Extracellular Regulated Kinase   FBS, Fetal Bovine Serum   FTI, Farnesyl Transferase Inhibitor   HFF, Human Foreskin Fibroblasts   HCMV, Human Cytomegalovirus   HPI, Hours Post-Infection   HHV, Human Herpes Virus   HSV, Herpes Simplex Virus   IE, Immediate Early   IPTG, isopropyl-1thio-β-D galactoside   L, Late   MAPK, Mitogen Activated Protein Kinases   MAPKK, MAPK kinase   MAPKKK, MAPK kinase kinase   MOI, Multiplicity of Infection   NF-κB, Nuclear Factor kappa light chain enhancer of activated B cells   PAA, Phosphono acetic Acid   PFU, Plaque-Forming Units   PI3-K, Phosphatidylinositol 3-Kinase   PKR, RNA-activated protein kinase   RALGDS, Ral Guanine Nucleotide Dissociation Stimulators   SDS, Sodium-Dodecyl Sulphate   TNF α, Tumor Necrosis Factor   VZV, Varicella Zoster Virus
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