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RECK suppresses interleukin-17/TRAF3IP2-mediated MMP-13 activation and human aortic smooth muscle cell migration and proliferation
Authors:Srinivas Mummidi  Nitin A Das  Andrea J Carpenter  Tadashi Yoshida  Manjunath Yariswamy  Ricardo Mostany  Reza Izadpanah  Yusuke Higashi  Sergiy Sukhanov  Makoto Noda  Ulrich Siebenlist  Randy S Rector  Bysani Chandrasekar
Institution:1. Department of Human Genetics, South Texas Diabetes and Obesity Institute, The University of Texas Rio Grande Valley School of Medicine, Edinburg, Texas;2. Department of Cardiothoracic Surgery, University of Texas Health Science Center, San Antonio, Texas;3. Department of Medicine/Cardiovascular Medicine, University of Missouri School of Medicine, Columbia, Missouri;4. Department of Pharmacology, Tulane University School of Medicine, New Orleans, Louisiana;5. Department of Medicine/Cardiology, Tulane University School of Medicine, New Orleans, Louisiana;6. Department of Molecular Oncology, Kyoto University Graduate School of Medicine, Kyoto, Japan;7. Laboratory of Immunoregulation, NIAID/NIH, Bethesda, Maryland;8. Research Service, Harry S. Truman Memorial Veterans Hospital, Columbia, Missouri

Department of Medicine/Gastroenterology and Hepatology/Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri

Abstract:
Keywords:inflammation  matrix metalloproteinases  neointima  RECK  restenosis
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