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The HDAC inhibitor SAHA does not rescue CFTR membrane expression in Cystic Fibrosis
Institution:1. Laboratory of Molecular Genetics, CHU Montpellier, Montpellier F-34093, France;2. EA7402, Rare Diseases Laboratory, University of Montpellier, Montpellier F-34093, France;3. Respiratory Disease Department – CHU Montpellier, Montpellier F-34295, France;4. PhyMedExp, University of Montpellier, INSERM U1046, CNRS UMR 9214, Montpellier F-34295, France;5. Service DIM, CHU Montpellier, Montpellier F-34093, France;1. Center for Cancer Biology, VIB, 3000 Leuven, Belgium;2. Laboratory of Translational Genetics, Department of Human Genetics, KU Leuven, 3000 Leuven, Belgium;1. Center for Translational Medicine, Department of Medicine, Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, PA 19107, USA;2. Aab Cardiovascular Research Institute, Department of Medicine, University of Rochester School of Medicine and Dentistry, Rochester, NY 14642, USA;3. From the Molecular Oncology Program, Lombardi Comprehensive Cancer Center, Georgetown University Medical Center, Washington, D. C. 20057,;4. Cancer Research Program, Houston Methodist Hospital Research Institute, Houston, Texas 77030, and;5. Department of Biochemistry and Molecular Medicine, The George Washington University, Washington, D. C. 20037;1. Department of Health, Animal Science and Food Safety, Università degli Studi di Milano, Via Celoria, 10 20133 Milan, Italy;2. Department of Animal Science, Oklahoma State University, Stillwater, OK 74078, USA
Abstract:
Keywords:Histone deacetylase inhibitor (HDACi)  Cystic fibrosis (CF)  Air-liquid interface cell culture  Cell differentiation
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