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MafA-deficient and beta cell-specific MafK-overexpressing hybrid transgenic mice develop human-like severe diabetic nephropathy
Authors:Homare Shimohata  Keigyou Yoh  Naoki Morito  Hiromi Tanaka  Masaki Kobayashi  Kunihiro Yamagata
Institution:a Pathophysiology of Renal Diseases, Doctoral Program in Clinical Sciences, Graduate School of Comprehensive Human Sciences, University of Tsukuba, Ibaraki, Japan
b Department of Anatomy and Embryology, Doctoral Program in Life System Medical Sciences, Graduate School of Comprehensive Human Sciences, University of Tsukuba, Ibaraki, Japan
c Department of Nephrology, Tokyo Medical University, Ibaraki Medical Center, Japan
Abstract:
Keywords:MafA  MafK  Diabetes mellitus  Diabetic nephropathy
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