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SARS-CoV-2 replication in airway epithelia requires motile cilia and microvillar reprogramming
Affiliation:1. Baxter Laboratory, Department of Microbiology & Immunology, Stanford University School of Medicine, Center for Clinical Sciences Research, 269 Campus Drive, Stanford, CA, USA;2. Department of Microbiology and Immunology, University of California, San Francisco, 600 16th Street, Room S572E, Box 2280, San Francisco, CA, USA;3. Department of Biology, Stanford University, Stanford, CA, USA;4. Department of Pathology, Stanford University School of Medicine, Stanford, CA, USA;5. Division of Allergy, Immunology, and Rheumatology, Department of Pediatrics, Stanford University School of Medicine, Stanford, CA, USA;6. Department of Otolaryngology–Head and Neck Surgery, Stanford University School of Medicine, Stanford, CA, USA;7. Department of Otorhinolaryngology, Jikei University School of Medicine, Tokyo, Japan;8. Department of Pediatric Pulmonary Medicine, Stanford University School of Medicine, Stanford, CA, USA;9. Disease Intervention and Prevention and Population Health Programs, Texas Biomedical Research Institute, San Antonio, TX, USA;10. Department of Chemistry, Stanford University, Stanford, CA, USA;11. Department of Otolaryngology, VA Palo Alto Health Care System, Palo Alto, CA, USA
Abstract:
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  • Keywords:SARS-CoV-2  COVID-19  organoids  airway epithelial cells  cilia  respiratory virus  proteomics  respiratory synctital virus  protein kinases  microvilli
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