| Abstract: | Glycine injection to the identified area of the RVLM leads to the development of a characteristic depressor reaction, due to a decrease of the total peripheral vascular resistance (TRP). Glycine injection to the identified area of CVLM results in the developing pressor reaction also due to the changes of TRP. Glycine injection to CVLM under preliminary functional switch-off the RVLM neuronal activity is not accompanied by the development of pressor reaction. This can be an evidence that CVLM sympathoinhibitory neurones act on cardiohemodynamics by their interaction with RVLM sympathoexcitatory neurones. Turn off of the tonically active sympathoinhibitory CVLM neurones does not affect the character of the reactions depending on the inhibition or excitation of sympathoexcitatory RVLM neurones. |
