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p205, a potential tumor suppressor, inhibits cell proliferation via multiple pathways of cell cycle regulation

Authors:	Asefa Benyam Dermott Jonathan M Kaldis Philipp Stefanisko Karen Garfinkel David J Keller Jonathan R

Institution:	Laboratory of Molecular Immunoregulation, Center for Cancer Research, National Cancer Institute-Frederick, Building 560, Room 31-56, Frederick, MD 21702-1201, USA.

Abstract:

Keywords:	IFI-200 interferon-inducible p200 family Cdk cyclin-dependent kinase CAK Cdk-activating kinase PCNA proliferating cell nuclear antigen GST glutathione-S-transferase 53BP p53-binding protein Miz1 c-Myc-interacting zinc-finger protein 1 AIM2 absent in melanoma 2 MNDA myeloid nuclear differentiation antigen DAPIN domain in apoptosis and interferon response PCR polymerase chain reaction DMEM Dulbecco’s modified Eagle’s medium PBS phosphate buffered saline BSA bovine serum albumin SDS-PAGE sodium dodecyl sulfate-polyacrylamide gel electrophoresis MPRO myeloid progenitor cell line
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