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Disruption of Acetyl-Lysine Turnover in Muscle Mitochondria Promotes Insulin Resistance and Redox Stress without Overt Respiratory Dysfunction
Affiliation:1. Duke Molecular Physiology Institute and Sarah W. Stedman Nutrition and Metabolism Center, Duke University Medical Center, Durham, NC 27701, USA;2. Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, TN 37232, USA;3. Vanderbilt Mouse Metabolic Phenotyping Center, Nashville, TN, USA;4. Department of Medicine, Division of Endocrinology, Metabolism Nutrition, Duke University Medical Center, Durham, NC 27701, USA;5. Department of Pharmacology & Cancer Biology, Duke University Medical Center, Durham, NC 27701, USA
Abstract:
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  • Keywords:sirtuins  lysine acetylation  proteomics  muscle  mitochondria  bioenergetics  fatty acid oxidation  insulin action  diabetes  obesity  nutrition  fat oxidation  respiration  redox  NAD biology  reactive oxygen species
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