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Glucose-induced CRL4COP1-p53 axis amplifies glycometabolism to drive tumorigenesis
Affiliation:1. Hechuan District People''s Hospital, Chongqing, China;2. Chongqing Key Research Laboratory of Drug Metabolism, College of Pharmacy, Chongqing Medical University, Chongqing, China;3. Institute of Life Sciences, Chongqing Medical University, Chongqing, China
Abstract:
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  • Keywords:glucose sensing  glycometabolism  p53 degradation  ubiquitylation  O-GlcNAcylation  CK2  CSN  overnutrition-associated cancer  neddylation
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