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An EMG-assisted model calibration technique that does not require MVCs
Authors:Jonathan S. Dufour  William S. Marras  Gregory G. Knapik
Affiliation:The Biodynamics Laboratory, The Ohio State University, 210 Baker Systems, 1971 Neil Avenue, Columbus, OH 43210, USA
Abstract:As personalized biologically-assisted models of the spine have evolved, the normalization of raw electromyographic (EMG) signals has become increasingly important. The traditional method of normalizing myoelectric signals, relative to measured maximum voluntary contractions (MVCs), is susceptible to error and is problematic for evaluating symptomatic low back pain (LBP) patients. Additionally, efforts to circumvent MVCs have not been validated during complex free-dynamic exertions. Therefore, the objective of this study was to develop an MVC-independent biologically-assisted model calibration technique that overcomes the limitations of previous normalization efforts, and to validate this technique over a variety of complex free-dynamic conditions including symmetrical and asymmetrical lifting. The newly developed technique (non-MVC) eliminates the need to collect MVCs by combining gain (maximum strength per unit area) and MVC into a single muscle property (gain ratio) that can be determined during model calibration. Ten subjects (five male, five female) were evaluated to compare gain ratio prediction variability, spinal load predictions, and model fidelity between the new non-MVC and established MVC-based model calibration techniques. The new non-MVC model calibration technique demonstrated at least as low gain ratio prediction variability, similar spinal loads, and similar model fidelity when compared to the MVC-based technique, indicating that it is a valid alternative to traditional MVC-based EMG normalization. Spinal loading for individuals who are unwilling or unable to produce reliable MVCs can now be evaluated. In particular, this technique will be valuable for evaluating symptomatic LBP patients, which may provide significant insight into the underlying nature of the LBP disorder.
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