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ATG5 is required to limit cell death induced by |
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| Authors: | Jingchun?Wang,Chao?Ma,Min?Zhang,Liu?Yang,Wenli?Chen author-information" > author-information__contact u-icon-before" > mailto:wenlichen@yahoo.com" title=" wenlichen@yahoo.com" itemprop=" email" data-track=" click" data-track-action=" Email author" data-track-label=" " >Email author |
| Affiliation: | 1.MOE Key Laboratory of Laser Life Science and Institute of Laser Life Science, College of Biophotonics,South China Normal University,Guangzhou,China;2.Guangdong Key Laboratory of Biotechnology for Plant Development, College of Life Science,South China Normal University,Guangzhou,China |
| Abstract: | Autophagy can be regarded as a protection mechanism to restrict programmed cell death (PCD) induced by pathogen infection during plant innate immunity in the early stages. Autophagy related 5 (ATG5) plays an important role in autophagy in Arabidopsis. We investigated the function of ATG5 in Arabidopsis in the hypersensitive response (HR)-PCD elicited by both virulent and avirulent strains of Pseudomonas syringae pv. tomato bacteria DC3000. Results show that ATG5 plays a vital role in limiting HR induced by P. syringae strains and colocalizes with autophagic bodies during the early phase of bacterial infection. In addition, the P. syringae-induced response is mediated by the salicylic acid (SA) signaling pathway. In summary, ATG5 is required for limiting HR-PCD induced in Arabidopsis by P. syringae strains and may be mediated by SA signaling. |
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