2',5'-Dihydroxychalcone-induced glutathione is mediated by oxidative stress and kinase signaling pathways期刊界 All Journals 搜尽天下杂志传播学术成果专业期刊搜索期刊信息化学术搜索

2',5'-Dihydroxychalcone-induced glutathione is mediated by oxidative stress and kinase signaling pathways

Authors:	Kachadourian Remy Pugazhenthi Subbiah Velmurugan Kalpana Backos Donald S Franklin Christopher C McCord Joe M Day Brian J

Affiliation:	^a Department of Medicine, National Jewish Health, Denver, CO 80206, USA ^b Department of Medicine, University of Colorado at Denver, Aurora, CO 80045, USA ^c Denver VA Medical Center, Denver, CO 80220, USA ^d Department of Pharmaceutical Sciences, University of Colorado at Denver, Aurora, CO 80045, USA

Abstract:

Keywords:	AP-1, activator protein-1 ARE, antioxidant-response element BCRP, breast cancer resistance protein BSO, smallcaps" >l-buthionine sulfoximine 2&prime ,5&prime -DHC, 2&prime ,5&prime -dihydroxychalcone ERK, extracellular-signal-regulated kinase GCLC, glutamate-cysteine ligase catalytic subunit GCLM, glutamate-cysteine ligase regulatory subunit GSH, reduced glutathione HO-1, heme oxygenase-1 2&prime -HC, 2&prime -hydroxychalcone HC, hydroxychalcone JNK, c-Jun N-terminal kinase LDH, lactate dehydrogenase MAPK, mitogen-activated protein kinase MnTDE-1,3-IP⁵⁺, manganese(III) meso-tetrakis(N,N&prime -diethylimidazolium-2-yl)porphyrin MRP, multidrug resistance-associated protein NAC, N-acetylcysteine PI3K, phosphatidylinositol 3-kinase ROS, reactive oxygen species Nrf2, NF-E2-related factor-2
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