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mTORC1 activation is not sufficient to suppress hepatic PPARα signaling or ketogenesis
Authors:Ebru S. Selen  Michael J. Wolfgang
Affiliation:1.Department of Biological Chemistry, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA;2.Department of Pharmacology and Molecular Sciences, The Johns Hopkins University School of Medicine, Baltimore, Maryland, USA
Abstract:
Keywords:metabolism, fatty acid oxidation, carnitine palmitoyltransferase 2 (Cpt2), peroxisome proliferator-activated receptor alpha (PPARα  ), mTOR, ketogenesis, β  -hydroxybutyrate (β  HB)
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