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Inhibition of PRMT3 activity reduces hepatic steatosis without altering atherosclerosis susceptibility in apoE knockout mice

Authors:	Menno Hoekstra Joya E Nahon Laura M de Jong Mara J Kröner Lidewij R de Leeuw Miranda Van Eck

Institution:	Division of BioTherapeutics, Leiden Academic Centre for Drug Research, Gorlaeus Laboratories, Einsteinweg 55, 2333CC Leiden, the Netherlands.

Abstract:

Keywords:	Corresponding author ABC transporter ATP-binding cassette transporter ACC1 acetyl-CoA carboxylase 1 ADMA asymmetric dimethylarginine ALT alanine aminotransferase ApoE apolipoprotein E cCIMT common Carotid Intima-Media Thickness FAS fatty acid synthase LPL lipoprotein lipase LXR liver X receptor MCP1 monocyte chemoattractant protein 1 oxLDL oxidized LDL PRMT3 protein arginine methyltransferase 3 SDMA symmetric dimethylarginine SREBP-1c sterol regulatory element-binding protein-1c SCD1 stearoyl-coenzyme A desaturase 1 TNFα tumor necrosis factor alpha UCP1 uncoupling protein 1 Protein arginine methyltransferase Atherosclerosis Fatty liver disease Hepatic steatosis Pharmacological inhibition Mouse model
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