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A Role of RIP3-Mediated Macrophage Necrosis in Atherosclerosis Development
Authors:Juan Lin  Hanjie Li  Min Yang  Junming Ren  Zhe Huang  Felicia Han  Jian Huang  Jianhui Ma  Duanwu Zhang  Zhirong Zhang  Jianfeng Wu  Deli Huang  Muzhen Qiao  Guanghui Jin  Qiao Wu  Yinghui Huang  Jie Du  Jiahuai Han
Affiliation:1. State Key Laboratory of Cellular Stress Biology and School of Life Sciences, Xiamen University, Xiamen, Fujian, China;2. Beijing Institute of Heart, Lung and Blood Vessel Diseases, Beijing Anzhen Hospital, Capital Medical University, Beijing 100029, China;3. The First Affiliated Hospital of Xiamen University, Xiamen, Fujian, China;4. Department of Basic Medical Sciences, Medical College, Xiamen University, Xiamen, Fujian, China;5. China-Japan Union Hospital, Jilin University, Changchun, Jilin, China
Abstract:
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  • Highlights? RIP3 deficiency in macrophages reduces advanced atherosclerotic lesions ? RIP3 deficiency reduces primary necrosis of macrophages in advanced lesions ? RIP3-dependent macrophage necrosis is not postapoptotic cell death ? Increased RIP3 converts apoptosis to necrosis in advanced lesional macrophages
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