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CCAT1/FABP5 promotes tumour progression through mediating fatty acid metabolism and stabilizing PI3K/AKT/mTOR signalling in lung adenocarcinoma
Authors:Jing Chen  Yaser Alduais  Kai Zhang  Xiaoli Zhu  Baoan Chen
Institution:1. Department of Hematology and Oncology, School of Medicine, Zhongda Hospital, Southeast University, Nanjing, China;2. Department of Hematology and Oncology, School of Medicine, Zhongda Hospital, Southeast University, Nanjing, China

Contribution: Data curation (lead), Writing - original draft (supporting);3. Department of Respiratory Medicine, Nanjing First Hospital, Nanjing, China

Contribution: Formal analysis (lead), ?Investigation (lead), Methodology (lead);4. Department of Respiratory Medicine, School of Medicine, Zhongda Hospital, Southeast University, Nanjing, China

Contribution: Writing - original draft (supporting), Writing - review & editing (supporting);5. Department of Hematology and Oncology, School of Medicine, Zhongda Hospital, Southeast University, Nanjing, China

Contribution: Project administration (equal), Writing - original draft (supporting), Writing - review & editing (supporting)

Abstract:
Keywords:angiogenesis  CCAT1  fatty acid metabolism  lung adenocarcinoma  ubiquitination
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