| 炎症与急性缺血性肾损伤 |
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| 引用本文： | 廖晓辉,张玲. 炎症与急性缺血性肾损伤[J]. 生命的化学, 2008, 28(6) |
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| 作者姓名： | 廖晓辉 张玲 |
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| 作者单位： | 重庆医科大学附属第二医院肾内科,重庆,400010;重庆医科大学附属第二医院肾内科,重庆,400010 |
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| 摘 要： | 近年来研究发现细胞间黏附分子-1和单核细胞趋化蛋白-1等炎症因子、核因子-κB及中性粒细胞、单核/巨噬细胞等炎症细胞参与了急性缺血性肾损伤的发生发展,抑制急性缺血性肾损伤时肾脏的炎症反应具有保护肾脏作用.
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| 关 键 词： | 急性缺血性肾损伤 缺血再灌注 炎症 |
The research progresses of inflammation and acute ischemic kidney injury |
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| Xiao-Hui LIAO,Ling ZHANG. The research progresses of inflammation and acute ischemic kidney injury[J]. Chemistry of Life, 2008, 28(6) |
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| Authors: | Xiao-Hui LIAO Ling ZHANG |
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| Affiliation: | Xiao-Hui LIAO Ling ZHANG (Chongqing University of Medical Science,Chongqing 400010,China) |
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| Abstract: | Investigations in recent years have confirmed that intercellular adhesion molecule-1, nuclear factor kappa B, monocyte chemoattractant protein-1, neutrophils, macrophages and lymphocytes were involved in the development of acute ischemic kidney injury (AIKI). Inhibition of renal inflammation could protect kidney from AIKI. |
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| Keywords: | acute ischemic kidney injury ischemia/reperfusion inflammation |
| 本文献已被 CNKI 维普 万方数据 等数据库收录！ |
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