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Role of sestrin2 in the regulation of proinflammatory signaling in macrophages
Institution:1. College of Pharmacy, Chosun University, Seosuk-dong, Dong-gu, Gwangju 501-759, South Korea;2. Department of Biochemistry, Chonbuk National University Medical School, Jeonju, Jeonbuk 561-756, Republic of Korea;3. MRC-GHF, College of Korean Medicine, Daegu Haany University, Gyeongsan, Gyeongsangbuk-do 712-715, Republic of Korea;1. Zhejiang Provincial Key Laboratory for Technology & Application of Model Organisms, School of Life Sciences, Wenzhou Medical University, Wenzhou, Zhejiang, China;2. Nelson Institute of Environmental Medicine, New York University School of Medicine, Tuxedo, NY, 10987, USA;3. State Key Laboratory of Phytochemistry and Plant Resources in West China, Kunming Institute of Botany, Chinese Academy of Sciences, Kunming, 650204, China;1. Affiliated Traditional Chinese Medicine Hospital of Xinjiang Medical University, Huanghe Road, Urumqi City, XinJiang Province, China;2. Guangdong Second Provincial General Hospital. Add: Xingangzhong Road, Guangzhou City, Guangdong Province, China;1. School of Biochemistry and Immunology, Trinity Biomedical Sciences Institute, Trinity College Dublin, Pearse Street, Dublin 2, Ireland;2. Center for Precision Genome Editing and Genetic Technologies for Biomedicine, Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, Moscow, Russia
Abstract:
Keywords:Sestrin2  TLR  Inflammation  Oxidative stress  JNK  AP-1
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