<Emphasis Type="Italic">Chlamydia pneumoniae</Emphasis> induces aponecrosis in human aortic smooth muscle cells |
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Authors: | Claudia?Dumrese Christine?F?Maurus Daniel?Gygi M?rten?KJ?Schneider Michael?Walch Peter?Groscurth Email author" target="_blank">Urs?ZieglerEmail author |
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Institution: | 1.Division of Cell Biology, Institute of Anatomy,University Zürich,Switzerland;2.Laboratory for Transplantation Immunology, Department of Internal Medicine,University Hospital Zürich,Switzerland |
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Abstract: | Background The intracellular bacterium Chlamydia pneumoniae is suspected to play a role in formation and progression of atherosclerosis. Many studies investigated cell death initiation
versus inhibition by Chlamydia pneumoniae in established cell lines but nothing is known in primary human aortic smooth muscle cells, a cell type among others known
to be involved in the formation of the atherosclerotic plaque. Type of cell death was analyzed by various methods in primary
aortic smooth muscle cells after infection with Chlamydia pneumoniae to investigate a possible pathogenic link in atherosclerosis. |
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