1. Department of Biochemistry and Molecular Biology, Johns Hopkins University Bloomberg School of Public Health, 615 North Wolfe Street, Baltimore, MD 21205, USA
Abstract:
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Highlights? Glucose and O2 signal through Cu/Zn SOD1 to repress respiration ? SOD1 binds to and prevents turnover of a casein kinase CK1γ for respiration control ? Superoxide from aerobic glucose metabolism feeds the SOD1 reaction to stabilize CK1γ ? Both human and yeast CK1γ stability are regulated by SOD1
