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SUCLA2-coupled regulation of GLS succinylation and activity counteracts oxidative stress in tumor cells
Authors:Yingying Tong  Dong Guo  Shu-Hai Lin  Jiazhen Liang  Dianqiang Yang  Chunmin Ma  Fei Shao  Min Li  Qiujing Yu  Yuhui Jiang  Lei Li  Jing Fang  Rilei Yu  Zhimin Lu
Affiliation:1. The Institute of Cell Metabolism and Diseases, Shanghai Key Laboratory of Pancreatic Disease, Shanghai General Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai 200080, China;2. Cancer Center, Beijing Luhe Hospital, Capital Medical University, Beijing 101149, China;3. Department of Hepatobiliary and Pancreatic Surgery and Zhejiang Provincial Key Laboratory of Pancreatic Disease of The First Affiliated Hospital, Institute of Translational Medicine, Zhejiang University School of Medicine, Hangzhou, Zhejiang 310029, China;4. Zhejiang University Cancer Center, Hangzhou, Zhejiang 310029, China;5. State Key Laboratory for Cellular Stress Biology, Innovation Center for Cell Signaling Network, School of Life Sciences, Xiamen University, Xiamen, Fujian 361102, China;6. Key Laboratory of Marine Drugs, Chinese Ministry of Education, School of Medicine and Pharmacy, Ocean University of China, Qingdao, Shandong 266003, China;7. The Affiliated Hospital of Qingdao University, Qingdao Cancer Institute, Qingdao, Shandong 266071, China;8. Key Laboratory of Immune Microenvironment and Disease (Ministry of Education), Department of Immunology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin 300070, China
Abstract:
Keywords:GLS  SUCLA2  p38  succinylation  phosphorylation  succinyl-CoA  NADPH  GSH  glutaminolysis  tumorigenesis
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