Factors controlling glycogen breakdown in the alligator |
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| Affiliation: | 1. Department of Biochemistry, LSU Medical Center, 1542 Tulane Avenue, New orleans, LA 70112, U.S.A.;2. Department of Pharmacology, LSU Medical Center, 1542 Tulane Avenue, New orleans, LA 70112, U.S.A.;1. Independent researcher, Amqui, Québec, Canada, G5J 2N5;2. Faculté de Médecine Vétérinaire, Université de Montréal, 3200, rue Sicotte, Saint-Hyacinthe, Québec, Canada, J2S 2M2;1. Nutrition and Endocrine Research Center, Research Institute for Endocrine Sciences, Shahid Beheshti University of Medical Sciences, Tehran, Iran;2. Prevention of Metabolic Disorders Research Center, Research Institute for Endocrine Sciences, Shahid Beheshti University of Medical Sciences, Iran;3. Endocrine Research Center, Research Institute for Endocrine Sciences, Shahid Beheshti University of Medical Sciences, Tehran, Iran;1. School of Earth and Environmental Sciences, Central University of Himachal Pradesh, Kangra, Himachal Pradesh, 176206, India;2. Department of Biotechnology, Babasaheb Bhimrao Ambedkar University, Vidya Vihar, Raebareli Road, Lucknow, Uttar Pradesh, 226025, India;1. The Mina and Everard Goodman Faculty of Life Sciences, Bar Ilan University, Ramat Gan, Israel;2. Zoology Department, Tel Aviv University, Tel Aviv, Israel |
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| Abstract: | 1. Catecholamines were injected into normal alligators and into others pre-treated with an α blocker (phenoxybenzamine) or a β blocker (propranol).2. Liver glycogenolysis after epinephrine, norepinephrine or isoproterenol was blocked by the β blocker, and muscle glycolysis after epinephrine or norepinephrine by an α blocker. Isoproterenol-induced glycolysis was blocked by the β blocker.3. Glycolysis proceeded 10 × as fast after work as after catecholamines and it could not be blocked.4. Work had little effect on blood glucose.5. It was concluded that glycolysis from work did not involve catecholamine stimulation. |
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