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Critical role of the transient activation of p38 MAPK in the etiology of skeletal muscle insulin resistance induced by low-level in vitro oxidant stress
Authors:Diamond-Stanic Maggie K  Marchionne Elizabeth M  Teachey Mary K  Durazo David E  Kim John S  Henriksen Erik J
Institution:aInnovative Drug Research Laboratories, Research Division, Kyowa Hakko Kirin Co., Ltd., 3-6-6 Asahimachi, Machida, Tokyo 194-8533, Japan;bDivision of Molecular and Cell Genetics, Department of Molecular and Cellular Biology, School of Life Sciences, Faculty of Medicine, Tottori University, 86 Nishi-cho, Yonago, Tottori 683-8503, Japan;cBiomedical Science, Institute of Regenerative Medicine and Bio-function, Graduate School of Medical Science, Tottori University, 86 Nishi-cho, Yonago, Tottori 683-8503, Japan;dChromosome Engineering Research Center, Tottori University, 86 Nishi-cho, Yonago, Tottori 683-8503, Japan
Abstract:
Keywords:Human artificial chromosome (HAC)  Multiple transgene expression  Primary cells  Erythropoietin (EPO)  Ubiquitin enhancer  Induced pluripotent stem cells (iPSCs)
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