| Abstract: | Evolution of the recombination system caused by antagonistic species interactions (host and parasite, for example) was studied. The genetic structure of host population as well as that of parasite is explicitly present in our models. The selection intensity depends on host's resistance and parasite's virulence, both controlled by polygenic systems. The rec-system dynamics was numerically studied using the genetic operators method. It is shown that high-recombination alleles of the rec-modificator can have a short-term selective advantage in both interacting populations simultaneously. |
