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Exosomal cellular prion protein drives fibrillization of amyloid beta and counteracts amyloid beta‐mediated neurotoxicity
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The marine diatoms Lauderia annulata and Synedra planctonicaharvested during exponential growth were exposed to differentdoses of u.v.-B (286, 439 and 710 J m2 d1) for2 days. Uptake of 15N-nitrate was estimated before, during andafter u.v.-B radiation over 2 days. Exposure to high levelsof u.v.-B (710 J m2 d1) caused irreversible damageat the second daily irradiance. Lauderia cells were less affectedby u.v.-B stress than Synedra cells. 15N-nitrate uptake wasreduced under u.v.-B irradiance but could be reactivated within1 day following exposure to a low dose (286 J m2 d1).Higher levels of u.v.-B radiation (710 J m2 d1)led to irreversible damage. The pattern of 15N-incorporationinto several amino acids of Lauderia varied after 2 days ofu.v.-B radiation. 15N enrichment of glutamine increased markedlyafter u.v.-B stress (717 J m2 d1) whereas I5Nexcess of aspartic acid was significantly reduced. Results arediscussed with reference to the u.v.-B damage of the nitratetransport system. 相似文献
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