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排序方式: 共有117条查询结果,搜索用时 0 毫秒
91.
Jean-Pierre Vacher Jérôme Chave Francesco Gentile Ficetola Guilhem Sommeria-Klein Shengli Tao Christophe Thébaud Michel Blanc Agustín Camacho José Cassimiro Timothy J. Colston Maël Dewynter Raffael Ernst Philippe Gaucher Jerriane Oliveira Gomes Rawien Jairam Philippe J. R. Kok Jucivaldo Dias Lima Quentin Martinez Christian Marty Brice P. Noonan Pedro M. Sales Nunes Paul Ouboter Renato Recoder Miguel Trefaut Rodrigues Andrew Snyder Sérgio Marques-Souza Antoine Fouquet 《Journal of Biogeography》2020,47(8):1781-1791
92.
Raffael Winkler und Lukas Jenni 《Journal of Ornithology》1987,128(2):243-246
Summary The occurrence of sectoral postjuvenile primary moult is confirmed for Sardinian Warbler, Greenfinch, Goldfinch and Red Crossbill and is newly described for White Wagtail, Siskin and Cirl Bunting. A characteristic feature for this type of moult seems to be that the primary coverts belonging to the renewed primaries are not at all or irregularly moulted. It is supposed that sectoral primary moult is provided in the moult programme of many passerines but is only manifested under special conditions. One of these is an early hatching date. 相似文献
93.
Dias Rosa Maria Tófoli Raffael Marcos da Silva João Carlos Barbosa Gomes Luiz Carlos Agostinho Angelo Antonio 《Aquatic Ecology》2022,56(3):877-889
Aquatic Ecology - Habitat complexity can substantially alter trophic relationships, such as competitive and predatory interactions, between fish species. This study aimed to evaluate how trophic... 相似文献
94.
The Kluyveromyces lactis zymocin complex kills Saccharomyces cerevisiae cells in a process that involves tRNA cleavage by its tRNAse gamma-toxin subunit. In contrast to the gamma-toxin mode of action, the early steps of the zymocin response are less well characterized. Here, we present high-dosage suppressors of zymocin that encode a putative Pkc1-related kinase (ISR1) and UDP-glucose pyrophosphorylase (UGPase) (UGP1). Anti-UGPase Western blots and GAL10 - ISR1 overexpression suggest that zymocin suppression correlates with overproduction of UGPase or Isr1. As judged from protection against exo-zymocin and unaltered sensitivity to endogenous gamma-toxin, high-copy ISR1 and UGP1 operate in early, nontarget steps of the zymocin pathway. Consistent with a recent report on in vitro phosphorylation of Isr1 and UGPase by the CDK Pho85, high-copy ISR1 and UGP1 suppression of zymocin is abolished in a pho85 null mutant lacking CDK activity of Pho85. Moreover, suppression requires UGPase enzyme activity, and ISR1 overexpression also protects against CFW, a chitin-interfering poison. Our data agree with roles for UGPase in cell wall biosynthetic processes and for Isr1 in Pkc1-related cell wall integrity. In sum, high-copy ISR1 and UGP1 cells affect early steps of the zymocin response and potentially prevent the lethal K. lactis killer complex from establishing cell surface recognition and/or contact. 相似文献
95.
Constance Mehlgarten Daniel Jablonowski Karin D. Breunig Michael J. R. Stark Raffael Schaffrath 《Molecular microbiology》2009,73(5):869-881
In yeast, the role for the Elongator complex in tRNA anticodon modification is affected by phosphorylation of Elongator subunit Elp1. Thus, hyperphosphorylation of Elp1 due to inactivation of protein phosphatase Sit4 correlates with Elongator-minus phenotypes including resistance towards zymocin, a tRNase cleaving anticodons of Elongator-dependent tRNAs. Here we show that zymocin resistance of casein kinase hrr25 mutants associates with hypophosphorylation of Elp1 and that nonsense suppression by the Elongator-dependent SUP4 tRNA is abolished in hrr25 or sit4 mutants. Thus changes that perturb the evenly balanced ratio between hyper- and hypophosphorylated Elp1 forms present in wild-type cells lead to Elongator inactivation. Antagonistic roles for Hrr25 and Sit4 in Elongator function are further supported by our data that Sit4 inactivation is capable of restoring both zymocin sensitivity and normal ratios between the two Elp1 forms in hrr25 mutants. Hrr25 binds to Elongator in a fashion dependent on Elongator partner Kti12. Like sit4 mutants, overexpression of Kti12 triggers Elp1 hyperphosphorylation. Intriguingly, this effect of Kti12 is blocked by hrr25 mutations, which also show enhanced binding of Kti12 to Elongator. Collectively, our data suggest that rather than directly targeting Elp1, the Hrr25 kinase indirectly affects Elp1 phosphorylation states through control of Sit4-dependent dephosphorylation of Elp1. 相似文献
96.
97.
We have isolated Chl a-Chl c-carotenoid binding proteins from the dinoflagellates Prorocentrum minimum and Heterocapsa pygmaea grown under high (500 mol m–2 s–1, HL) and low (35 mol m–2 s–1, LL) light conditions. We compared various isolation procedures of membrane bound light harvesting complexes (LHCs) and assayed the functionality of the solubilized proteins by determining the energy transfer efficiency from the accessory pigments to Chl a by means of fluorescence excitation spectra. The identity of the newly isolated protein-complexes were confirmed by immunological cross-reactions with antibodies raised against the previously described membrane bound Chl a-c proteins (Boczar et al. (1980) FEBS Lett 120: 243–247). Spectroscopic analysis demonstrated the relatedness of these proteins with the recently described Chl-a-c
2-peridinin (ACP) binding protein (Hiller et al. (1993) Photochem Photobiol 57: 125–131; Iglesias Prieto et al. (1993) Phil Trans R Soc London B 338: 381–392). The water-soluble peridinin-Chl a binding-protein (PCP) was not detectable in P. minimum. Two functional forms of ACP with different pigmentation were isolated. A variant of ACP which was isolated from high-light grown cells, that specifically binds increased amounts of diadinoxanthin was compared to the previously described ACPs that bind proportionately more peridinin.Abbreviations ACP
Chl a-Chl c-peridinin binding protein
- AEBSF
4-(2-aminoethyl)-benzenesulfonyl fluoride hydrochloride
- DDM
dodecyl -d maltoside
- Deriphat 160
N-lauryl-beta-iminopropionic acid
- HEPES
(N-2-hydroxyethylpiparizine-N-2-ethanesulphonic acid)
- HL
high light (500 mol m–2 s–1)
- LL
low light (35 mol m–2 s–1)
- 730
fluorescence yield (emission at 730 nm)
- PCP
peridinin-Chl a-binding protein
- PMSF
phenyl-methyl-sulfonyl-fluoride
- PS I
Photosystem I
- PS II
Photosystem II 相似文献
98.
99.
100.
Priming: getting ready for battle 总被引:1,自引:0,他引:1
Prime-A-Plant Group Conrath U Beckers GJ Flors V García-Agustín P Jakab G Mauch F Newman MA Pieterse CM Poinssot B Pozo MJ Pugin A Schaffrath U Ton J Wendehenne D Zimmerli L Mauch-Mani B 《Molecular plant-microbe interactions : MPMI》2006,19(10):1062-1071
Infection of plants by necrotizing pathogens or colonization of plant roots with certain beneficial microbes causes the induction of a unique physiological state called "priming." The primed state can also be induced by treatment of plants with various natural and synthetic compounds. Primed plants display either faster, stronger, or both activation of the various cellular defense responses that are induced following attack by either pathogens or insects or in response to abiotic stress. Although the phenomenon has been known for decades, most progress in our understanding of priming has been made over the past few years. Here, we summarize the current knowledge of priming in various induced-resistance phenomena in plants. 相似文献