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61.
Ethan Fechter‐Leggett Brittany M. Nelms Christopher M. Barker William K. Reisen 《Journal of vector ecology》2012,37(2):442-449
West Nile virus (WNV) is now endemic in California, with annual transmission documented by the statewide surveillance system. Although much is known about the horizontal avian‐mosquito transmission cycle, less is known about vertical transmission under field conditions, which may supplement virus amplification during summer and provide a mechanism to infect overwintering female mosquitoes during fall. The current study identified clusters of WNV‐infected mosquitoes in Sacramento and Yolo Counties, CA, during late summer 2011 and tested field‐captured ovipositing female mosquitoes and their progeny for WNV RNA to estimate the frequency of vertical transmission. Space‐time clustering of WNV‐positive Culex pipiens complex pools was detected in the northern Elk Grove area of Sacramento County between July 18 and September 18, 2011 (5.22 km radius; p<0.001 and RR=7.80). Vertical transmission by WNV‐infected females to egg rafts was 50% and to larvae was 40%. The estimated minimal filial infection rate from WNV‐positive, ovipositing females was 2.0 infected females/1,000. The potential contribution of vertical transmission to WNV maintenance and amplification are discussed. 相似文献
62.
Keats Nelms Thomas J. O'Neill Shiqing Li Stevan R. Hubbard Thomas A. Gustafson William E. Paul 《Mammalian genome》1999,10(12):1160-1167
The SH2-B protein is an SH2-domain-containing molecule that interacts with a number of phosphorylated kinase and receptor
molecules including the insulin receptor. Two isoforms of the SH2-B have been identified and have been proposed to arise through
alternate splicing. Here we have identified a third isoform of the SH2-B protein, SH2-Bγ, that interacts specifically with
the insulin receptor. This interaction required phosphorylation of residue Y1146 in the triple tyrosine motif within the activation
loop of the IR kinase and is one of only two signaling molecules shown to interact directly with this residue of the insulin
receptor kinase domain. The intron/exon structure of the SH2-B gene was determined. Alternate splice sites utilized to generate
the different isoforms of the SH2-B protein were identified in the 3′ end of the SH2-B gene immediately downstream of the
exon encoding the core of the SH2 domain. Additionally, the chromosomal location of the SH2-B gene was determined to be the
distal arm of mouse Chromosome (Chr) 7 in a region linked to obesity in mice.
Received: 13 May 1999 / Accepted: 13 August 1999 相似文献
63.