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701.
Ling Huang Ridhdhi Desai Daniel N. Conrad Nayara C. Leite Dipikaa Akshinthala Christine Maria Lim Raul Gonzalez Lakshmi B. Muthuswamy Zev Gartner Senthil K. Muthuswamy 《Cell Stem Cell》2021,28(6):1090-1104.e6
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702.
Chandramohan Ramasamy Rahul Ramteke Jayaraman Balachander Raja J. Selvaraj 《Indian pacing and electrophysiology journal》2013,13(4):148-150
We present an interesting image showing sequential loss of anterograde, and subsequently, retrograde conduction during radiofrequency ablation of an accessory pathway. We discuss the possible mechanisms and prior literature concerning this interesting finding. 相似文献
703.
Jayaraman Selvaraj Rajagopal Ponnulakshmi Veeraraghavan Vishnupriya Chandrasekar Kirubhanand 《Bioinformation》2020,16(10):731
Staphylococcal enterotoxins (SEs) are liked with food poisoning and other related infections. Nafcillin is an antibiotic used to treat S. aureus. Therefore, it is of interest to study the molecular interactions of 25 nafcillin analogues with enterotoxin I using molecular docking analysis. The analysis shows optimal interaction features of Nafcillin analogues with Enterotoxin I from Staphylococcus aureus for further consideration. 相似文献
704.
Jay Jayaraman Minsoo Yoon Emma R. Applegate Erin A. Stroud Matthew D. Templeton 《Molecular Plant Pathology》2020,21(11):1467-1480
Pseudomonas syringae pv. actinidiae ICMP 18884 biovar 3 (Psa3) produces necrotic lesions during infection of its kiwifruit host. Bacterial growth in planta and lesion formation are dependent upon a functional type III secretion system (T3S), which translocates multiple effector proteins into host cells. Associated with the T3S locus is the conserved effector locus (CEL), which has been characterized and shown to be essential for the full virulence in other P. syringae pathovars. Two effectors at the CEL, hopM1 and avrE1, as well as an avrE1-related non-CEL effector, hopR1, have been shown to be redundant in the model pathogen P. syringae pv. tomato DC3000 (Pto), a close relative of Psa. However, it is not known whether CEL-related effectors are required for Psa pathogenicity. The Psa3 allele of hopM1, and its associated chaperone, shcM, have diverged significantly from their orthologs in Pto. Furthermore, the CEL effector hopAA1-1, as well as a related non-CEL effector, hopAA1-2, have both been pseudogenized. We have shown that HopM1 does not contribute to Psa3 virulence due to a truncation in shcM, a truncation conserved in the Psa lineage, probably due to the need to evade HopM1-triggered immunity in kiwifruit. We characterized the virulence contribution of CEL and related effectors in Psa3 and found that only avrE1 and hopR1, additively, are required for in planta growth and lesion production. This is unlike the redundancy described for these effectors in Pto and indicates that these two Psa3 genes are key determinants essential for kiwifruit bacterial canker disease. 相似文献
705.