全文获取类型
收费全文 | 21428篇 |
免费 | 1500篇 |
国内免费 | 28篇 |
出版年
2024年 | 27篇 |
2023年 | 69篇 |
2022年 | 243篇 |
2021年 | 415篇 |
2020年 | 249篇 |
2019年 | 312篇 |
2018年 | 531篇 |
2017年 | 394篇 |
2016年 | 684篇 |
2015年 | 1131篇 |
2014年 | 1229篇 |
2013年 | 1398篇 |
2012年 | 1834篇 |
2011年 | 1714篇 |
2010年 | 1101篇 |
2009年 | 917篇 |
2008年 | 1360篇 |
2007年 | 1189篇 |
2006年 | 1063篇 |
2005年 | 980篇 |
2004年 | 967篇 |
2003年 | 781篇 |
2002年 | 785篇 |
2001年 | 627篇 |
2000年 | 633篇 |
1999年 | 423篇 |
1998年 | 168篇 |
1997年 | 129篇 |
1996年 | 120篇 |
1995年 | 87篇 |
1994年 | 82篇 |
1993年 | 69篇 |
1992年 | 158篇 |
1991年 | 125篇 |
1990年 | 88篇 |
1989年 | 103篇 |
1988年 | 71篇 |
1987年 | 65篇 |
1986年 | 69篇 |
1985年 | 53篇 |
1984年 | 47篇 |
1983年 | 37篇 |
1982年 | 27篇 |
1981年 | 24篇 |
1978年 | 28篇 |
1976年 | 32篇 |
1975年 | 29篇 |
1973年 | 33篇 |
1971年 | 23篇 |
1969年 | 24篇 |
排序方式: 共有10000条查询结果,搜索用时 41 毫秒
771.
772.
773.
774.
Transition from Diffusion‐Controlled Intercalation into Extrinsically Pseudocapacitive Charge Storage of MoS2 by Nanoscale Heterostructuring 下载免费PDF全文
Qasim Mahmood Sul Ki Park Kideok D. Kwon Sung‐Jin Chang Jin‐Yong Hong Guozhen Shen Young Mee Jung Tae Jung Park Sung Woon Khang Woo Sik Kim Jing Kong Ho Seok Park 《Liver Transplantation》2016,6(1)
2D nanomaterials have been found to show surface‐dominant phenomena and understanding this behavior is crucial for establishing a relationship between a material's structure and its properties. Here, the transition of molybdenum disulfide (MoS2) from a diffusion‐controlled intercalation to an emergent surface redox capacitive behavior is demonstrated. The ultrafast pseudocapacitive behavior of MoS2 becomes more prominent when the layered MoS2 is downscaled into nanometric sheets and hybridized with reduced graphene oxide (RGO). This extrinsic behavior of the 2D hybrid is promoted by the fast Faradaic charge‐transfer kinetics at the interface. The heterostructure of the 2D hybrid, as observed via high‐angle annular dark field–scanning transmission electron microscopy and Raman mapping, with a 1T MoS2 phase at the interface and a 2H phase in the bulk is associated with the synergizing capacitive performance. This 1T phase is stabilized by the interactions with the RGO. These results provide fundamental insights into the surface effects of 2D hetero‐nanosheets on emergent electrochemical properties. 相似文献
775.
776.
777.
778.
Changhyun Choi Sangryeol Park Ilpyung Ahn Shinchul Bae Duk-Ju Hwang 《Plant biotechnology reports》2016,10(5):301-307
Soft rot caused by Pectobacterium carotovorum subsp. carotovorum (Pcc) is a serious disease in Chinese cabbage (Brassica rapa L. subsp. pekinensis). To reduce the severity of soft rot symptoms in Chinese cabbage, Arabidopsis AtWRKY75 was introduced into Chinese cabbage by Agrobacterium-mediated transformation, which was previously reported to reduce susceptibility to Pcc infection in Arabidopsis. Three independent Chinese cabbage transgenic lines carrying AtWRKY75 were obtained. The growth phenotypes of AtWRKY75 overexpression (OE) lines were normal. Bacterial soft rot symptoms and Pcc growth were reduced in AtWRKY75-OE Chinese cabbage lines compared with WT plants. In contrast, overexpression of AtWRKY75 had no effect on infection with a hemibiotrophic pathogen, Xanthomonas campestris pv. campestris (Xcc) causing black rot disease. These results are consistent with those observed in the transgenic Arabidopsis. We found that AtWRKY75 activated a subset of Chinese cabbage genes related to defense against Pcc infection, such as Meri15B, BrPR4, and BrPDF1.2 (but not BrPGIP2). Moreover, overexpression of AtWRKY75 caused H2O2 production and activation of H2O2 scavenge enzyme genes, suggesting that H2O2 played a role in AtWRKY75-mediated resistance to Pcc. Together, these results demonstrated that AtWRKY75 decreased the severity of Pcc-caused bacterial soft rot and activated a subset of Pcc infection defense-related genes in Chinese cabbage similar to in Arabidopsis. It is suggested that AtWRKY75 is a candidate gene for use in crop improvement, because it results in reduced severity of disease symptoms without concurrent growth abnormalities. 相似文献
779.
780.
Andrea Santeford Luke A. Wiley Sunmin Park Sonya Bamba Rei Nakamura Abdelaziz Gdoura 《Autophagy》2016,12(10):1876-1885
Autophagy is critical for maintaining cellular homeostasis. Organs such as the eye and brain are immunologically privileged. Here, we demonstrate that autophagy is essential for maintaining ocular immune privilege. Deletion of multiple autophagy genes in macrophages leads to an inflammation-mediated eye disease called uveitis that can cause blindness. Loss of autophagy activates inflammasome-mediated IL1B secretion that increases disease severity. Inhibition of caspase activity by gene deletion or pharmacological means completely reverses the disease phenotype. Of interest, experimental uveitis was also increased in a model of Crohn disease, a systemic autoimmune disease in which patients often develop uveitis, offering a potential mechanistic link between macrophage autophagy and systemic disease. These findings directly implicate the homeostatic process of autophagy in blinding eye disease and identify novel pathways for therapeutic intervention in uveitis. 相似文献