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991.
Qiang‐Qiang Xiong Tian‐Hua Shen Lei Zhong Chang‐Lan Zhu Xiao‐Song Peng Xiao‐Peng He Jun‐Ru Fu Lin‐Juan Ouyang Jian‐Min Bian Li‐Fang Hu Xiao‐Tang Sun Jie Xu Hui‐Ying Zhou Hao‐Hua He Xiao‐Rong Chen 《Physiologia plantarum》2019,167(4):564-584
Abrupt drought–flood alternation (T1) is a meteorological disaster that frequently occurs during summer in southern China and the Yangtze river basin, often causing a significant loss of rice production. In this study, the response mechanism of yield decline under abrupt drought–flood alternation stress at the panicle differentiation stage was analyzed by looking at the metabolome, proteome as well as yield and physiological and biochemical indexes. The results showed that drought and flood stress caused a decrease in the yield of rice at the panicle differentiation stage, and abrupt drought–flood alternation stress created a synergistic effect for the reduction of yield. The main reason for the decrease of yield per plant under abrupt drought–flood alternation was the decrease of seed setting rate. Compared with CK0 (no drought and no flood), the net photosynthetic rate and soluble sugar content of T1 decreased significantly and its hydrogen peroxidase, superoxide dismutase, peroxidase activity increased significantly. The identified differential metabolites and differentially expressed proteins indicated that photosynthesis metabolism, energy metabolism pathway and reactive oxygen species response have changed strongly under abrupt drought–flood alteration stress, which are factors that leads to the rice grain yield reduction. 相似文献
992.
Plant and Soil - Iron plaque at the soil-root interface is a key position for uptake of heavy metals by plants. Exploring iron plaque’s role in Pb uptake by edible vegetable roots aids... 相似文献
993.
Paecilomyces variotii extracts (ZNC) enhance plant immunity and promote plant growth 总被引:1,自引:0,他引:1
Lu Chongchong Liu Haifeng Jiang Depeng Wang Lulu Jiang Yanke Tang Shuya Hou Xuwen Han Xinyi Liu Zhiguang Zhang Min Chu Zhaohui Ding Xinhua 《Plant and Soil》2019,445(1-2):383-396
Plant and Soil - There are growing concerns regarding the restoration of karst rocky desertification (KRD) areas. However, the soil conditions and its residing microorganisms, which are essential... 相似文献
994.
995.
Zhou Yang Fu Yu Bai Zhendong Li Peixin Zhao Bo Han Yuehua Xu Ting Zhang Ningyan Lin Lin Cheng Jian Zhang Jun Zhang Jing 《Biological trace element research》2019,187(1):172-180
Biological Trace Element Research - The purpose of this study was to evaluate the protective effect of Du-Zhong cortex extract (DZCE) on lead acetate-induced bone loss in rats. Forty female... 相似文献
996.
Liu Siqi Wang Bo Li Xiaojing Pan Jingxian Qian Xuexue Yu Yahui Xu Ping Zhu Jian Xu Xiaofeng 《Plant Cell, Tissue and Organ Culture》2019,137(3):485-494
Plant Cell, Tissue and Organ Culture (PCTOC) - Callus is a remarkable regeneration tissue. The genes correlated with root development can be involved in regulating callus development in higher... 相似文献
997.
Long Huiping Zhong Gang Wang Chengzhi Zhang Jian Zhang Yueling Luo Jinglian Shi Shengliang 《Neurochemical research》2019,44(8):1830-1839
Neurochemical Research - The pathogenesis of late-onset Alzheimer's disease (LOAD) mainly involves abnormal accumulation of extracellular β-amyloid (Aβ) and the consequent neurotoxic... 相似文献
998.
Xiang-Lan Sun Sarah J. Lessard Ding An Ho-Jin Koh Hiroyasu Esumi Michael F. Hirshman Laurie J. Goodyear 《Journal of cellular biochemistry》2019,120(1):685-696
The signaling mechanisms mediating myocardial glucose transport are not fully understood. Sucrose nonfermenting AMP-activated protein kinase (AMPK)-related kinase (SNARK) is an AMPK-related protein kinase that is expressed in the heart and has been implicated in contraction-stimulated glucose transport in mouse skeletal muscle. We first determined if SNARK is phosphorylated on Thr208, a site critical for SNARK activity. Mice were treated with exercise, ischemia, submaximal insulin, or maximal insulin. Treadmill exercise slightly, but significantly increased SNARK Thr208 phosphorylation. Ischemia also increased SNARK Thr208 phosphorylation, but there was no effect of submaximal or maximal insulin. HL1 cardiomyocytes were used to overexpress wild-type (WT) SNARK and to knockdown endogenous SNARK. Overexpression of WT SNARK had no effect on ischemia-stimulated glucose transport; however, SNARK knockdown significantly decreased ischemia-stimulated glucose transport. SNARK overexpression or knockdown did not alter insulin-stimulated glucose transport or glycogen concentrations. To study SNARK function in vivo, SNARK heterozygous knockout mice (SNARK+/−) and WT littermates performed treadmill exercise. Exercise-stimulated glucose transport was decreased by ~50% in hearts from SNARK+/− mice. In summary, exercise and ischemia increase SNARK Thr208 phosphorylation in the heart and SNARK regulates exercise-stimulated and ischemia-stimulated glucose transport. SNARK is a novel mediator of insulin-independent glucose transport in the heart. 相似文献
999.
Bolun Cheng Xiao Liang Yan Wen Ping Li Lu Zhang Mei Ma Shiqiang Cheng Yanan Du Li Liu Miao Ding Yan Zhao Feng Zhang 《Journal of cellular biochemistry》2019,120(9):14831-14837
1000.
Yao Huang Wei Mei Jian Chen Tao Jiang Zheng Zhou Guoyong Yin Jin Fan 《Journal of cellular biochemistry》2019,120(2):1903-1915
In this experiment, the cross-talk betweenNotch and the NF-κB signaling pathway was examined to reveal the mechanism of slowing down the type II collagen (ColII) and aggrecan degeneration affected by inflammatory cytokines. The expression levels of ColII and aggrecan in the intervertebral disc were observed through immunohistochemistry and hematoxylin-eosin staining+alcian blue staining, respectively. The expression levels of ColII, aggrecan, Runx2, and NF-κB in the nuclei of human nucleus pulposus cells (hNPCs) in each group, as well as the phosphorylation and acetylation levels of p65, were examined through Western blot analysis. The 293T cells were transfected with a plasmid containing the overexpressed relative domain of Notch1 intracellular domain (NICD1), and immunoprecipitation (IP) was performed to observe the combination of NICD1 and p65. HNPCs were transfected with a lentiviral-contained overexpression lacking the ANK region of NICD1, and IP was performed to observe the combination of NICD1 and p65. The expression of ColII and aggrecan in the intervertebral disc culture increased when γ-secretase inhibitor N-[N-(3,5-difluorophenacetyl)-1-alanyl]-Sphenylglycine t-butyl ester (DAPT) was added to the disc culture medium. Western blot revealed that DAPT inhibited p65 phosphorylation and acetylation, and the p65 and p50 levels in the nucleus decreased. NICD1 was found to be combined with p65 in contrast to the reverse consequences after ANK domain deletion in hNPCs. In nucleus pulposus cells, the combination of p65 and the ANK domain of NICD1 is a critical procedure for the degeneration related to the NF-κB signaling pathway activation induced by IL-1β and TNF-α. 相似文献