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排序方式: 共有928条查询结果,搜索用时 15 毫秒
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Kyle B. Heine Wendy R. Hood 《Biological reviews of the Cambridge Philosophical Society》2020,95(3):730-737
We have a limited understanding of the proximate mechanisms that are responsible for the development of variation in animal performance and life‐history strategies. Provided that components of an organism's successful life history – for example, mate competition, gestation, lactation, etc. – are energetically demanding, increased energy production within mitochondria is likely the foundation from which organisms are able to perform these tasks. Mitochondrial behaviour (positioning within the cell and communication between mitochondria) and morphology affect variation in energy production at the molecular, cellular, and organismal levels. Therefore, adaptations in mitochondrial behaviour and morphology that favour efficient energy production likely influence variation in animal performance. Previous work has linked greater proportions of inter‐mitochondrial junctions and density of the inner mitochondrial membrane, among other traits, with increased energetic demand. Future research should focus on how inter‐mitochondrial junctions and morphology of the inner mitochondrial membrane, in particular, influence animal performance in accordance with mitochondrial density, fission, and fusion. 相似文献
923.
Two cases of basal-cell adenoma (basaloid monomorphic adenoma, dermal analogue tumor) seen in a one-year period were subjected to fine needle aspiration biopsy and subsequent histologic confirmation. Two cases of adenoid cystic carcinoma occurring in the parotid region were selected for comparison; a great similarity between the two types of tumors was noted. On aspiration smear both lesions consisted of clusters and sheets of uniform, small, regular cells with scant basophilic cytoplasm and regular round or oval bland nuclei. The smears contained varying amounts of homogeneous, pale, eosinophilic stromal substance within and around the cell clusters. Individual cell differences between the two lesions were minimal, with the adenoid cystic carcinomas tending to exhibit more nuclear hyperchromatism and irregularity; the architectural arrangements of the cell clusters and the stromal substance permitted differentiation between the two lesions. 相似文献
924.
J E Hood 《BMJ (Clinical research ed.)》1991,303(6808):995-996
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The ultimate goal of quantitative trait locus (QTL) mapping is to identify the genes affecting complex traits. Using animal
models, we recently identified QTLs on mouse Chromosomes (Chrs) 1, 4, and 11 affecting genetic predisposition to acute alcohol
withdrawal. Among mice derived from the C57BL/6J (B6) and DBA/2J (D2) inbred strains, the locus identified on Chr 11 (∼20
cM) accounted for 12% of the genetic variability in withdrawal liability. Candidate genes in proximity to this QTL encode
the γ2, α1, and α6 subunits of GABAA receptors. The present studies identify a polymorphism between the B6 and D2 strains in the γ2 subunit gene, Gabrg2, and expand genotypic analysis to their BXD recombinant inbred strains. This polymorphism predicts a difference in amino acid
sequence (Ala-11 vs. Thr-11) within the extracellular amino-terminal region of the γ2 subunit. Analysis using BXD strain means for acute alcohol withdrawal severity suggests that the γ2 subunit polymorphism is genetically correlated with alcohol withdrawal severity. This is the first report that QTL mapping
for an alcohol-related trait has successfully led to the identification of a polymorphic candidate gene product that is genetically
associated with the trait.
Received: 15 September 1998 / Accepted: 8 October 1998 相似文献
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