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941.
942.
John Patrick O'Grady Gerald N. Esra Caroline H. Yeager Warren D. Thomas 《Zoo biology》1982,1(2):135-140
Reproductive failure in two female gorillas in the Los Angeles Zoo led to evaluation for secondary infertility. Male factor was excluded by noting routine conceptions in other individuals in the same group, and by behavioral observations of coital activity with the infertile females. Biochemical evidence of ovarian function was obtained by urinary sex steroid analysis. Continuity of the reproductive tract of the infertile apes was then studied by hysterosalpingography and laparoscopy while their general health was evaluated by ultrasonography and physical examination. Progressive, multiorgan involvement with Echinococcus vogeli was documented and mebendazole therapy begun. The investigation of secondary infertility in great apes by combined methods is safe and accurate, but requires special equipment and experienced personnel. 相似文献
943.
944.
945.
Luara Isabela dos Santos Bruno Galvão-Filho Paula Cristina de Faria Caroline Junqueira Miriam Santos Dutra Santuza Maria Ribeiro Teixeira Maurício Martins Rodrigues Gerd Ritter Oliver Bannard Douglas Thomas Fearon Lis Ribeiro Antonelli Ricardo Tostes Gazzinelli 《Cancer immunology, immunotherapy : CII》2015,64(3):311-323
946.
947.
In agricultural areas, Aspergillus
flavus, Aspergillus fumigatus and Aspergillus parasiticus are commonly identified in various feedstuffs and bioaerosols originated from feed handling. Some isolates belonging to these
fungal species could produce mycotoxins and constitute a risk factor for human and animal health. In this study, Fourier-transform
infrared spectroscopy was used for a rapid detection and characterization of 99 isolates collected from agricultural areas.
The results showed a first cluster corresponding to strains previously attributed to the A.
fumigatus group according to current taxonomic concepts, and a second cluster divided in 2 groups around reference strains of A.
flavus and A. parasiticus species. The toxigenic capacity of isolates was evaluated by high performance liquid chromatography coupled to mass spectrometry.
In the A. flavus group, only 6 strains of A. parasiticus and 4 strains of A. flavus were able to produce aflatoxins on culture media. FT-IR spectroscopy, respectively, allowed the differentiation of non-toxigenic
and toxigenic A. flavus and A. parasiticus isolates at 75 and 100%. Discrimination between toxigenic and non-toxigenic A. fumigatus was not possible because all of the isolates produced at least one mycotoxin. 相似文献
948.
949.
Sydney X. Lu Emma De Neef James D. Thomas Erich Sabio Benoit Rousseau Mathieu Gigoux David A. Knorr Benjamin Greenbaum Yuval Elhanati Simon J. Hogg Andrew Chow Arnab Ghosh Abigail Xie Dmitriy Zamarin Daniel Cui Caroline Erickson Michael Singer Hana Cho Robert K. Bradley 《Cell》2021,184(15):4032-4047.e31
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950.
Hans Carl Hasselbalch Vibe Skov Thomas Stauffer Larsen Mads Thomassen Caroline Hasselbalch Riley Morten K. Jensen Ole Weis Bjerrum Torben A. Kruse 《PloS one》2014,9(1)
Identifying a distinct gene signature for myelofibrosis may yield novel information of the genes, which are responsible for progression of essential thrombocythemia and polycythemia vera towards myelofibrosis. We aimed at identifying a simple gene signature – composed of a few genes - which were selectively and highly deregulated in myelofibrosis patients. Gene expression microarray studies have been performed on whole blood from 69 patients with myeloproliferative neoplasms. Amongst the top-20 of the most upregulated genes in PMF compared to controls, we identified 5 genes (DEFA4, ELA2, OLFM4, CTSG, and AZU1), which were highly significantly deregulated in PMF only. None of these genes were significantly regulated in ET and PV patients. However, hierarchical cluster analysis showed that these genes were also highly expressed in a subset of patients with ET (n = 1) and PV (n = 4) transforming towards myelofibrosis and/or being featured by an aggressive phenotype. We have identified a simple 5-gene signature, which is uniquely and highly significantly deregulated in patients in transitional stages of ET and PV towards myelofibrosis and in patients with PMF only. Some of these genes are considered to be responsible for the derangement of bone marrow stroma in myelofibrosis. Accordingly, this gene-signature may reflect key processes in the pathogenesis and pathophysiology of myelofibrosis development. 相似文献