中国中气门螨新纪录(蜱螨亚纲:革螨股)

报道中国中气门螨8新纪录种.     

尾卵螨属在中国的发现并记述一新种(蜱螨亚纲:尾足螨股)

记述尾卵螨属l新种：安氏尾卵螨Uroobovella anwenjui sp．nov．,该属在中国为新纪录。模式标本存于全国鼠疫布氏菌病防治基地,吉林省白城市。     

具刺新革螨在中国的首次发现及其雄螨的描述：蜱螨亚纲：革螨股：寄螨科

描述具刺新划螨Neogamasus belemnophorus Athias-Henriot,1977雄螨,该种为中国新纪录     

中国枝厉螨属1新种和2新纪录（蜱螨亚纲：草螨股：胭螨科）

报道中国枝厉螨属1新种和2新纪录,新种为白氏枝厉螨Dendrolaelaps baix-uelii sp.nov.,新纪录为小坑枝厉螨Dendrolaelaps foveolatus(Leitner,1949)和沙生枝厉螨Dendrolaelaps arenarius Karg,1971。     

峰革赛螨幼若螨描述（蜱螨亚纲：革螨股：胭螨科）

在所查到的献中，峰革赛螨Gamasellus montanus(Willmann，1936)仅描述了雌和雄螨(BpereToBa和Щep5aK，1977)，本描述其后若螨、前若螨和幼螨。中测量单位为μm，括号内为测量均值。     

寄螨属四种前若螨描述（蜱螨亚纲：革螨股）

本文描述寄螨属4种前著螨;邓氏寄螨Parasitus tengkuofani Ma,1995,王氏寄螨parasitus wangdunqingi Ma,1995,温氏寄螨parasitus wentinghuani Ma,1996,二刺寄螨parasitus bis pinatus Ma,1996.     

厉螨科二新种（蜱螨亚纲：革螨股）

本文绘图描述了厉螨科二新种:拟单阳厉螨Androlaelaps singuloides sp.nov.与陇川下盾螨Hypoaspis longchuanensis sp.nov.,标本分别采自蟋蟀与独角仙体上,采集地同为云南陇川县,文中还列举了它们与相近种类的鉴别要点。     

饰样小全盾螨雄螨描述（蜱螨亚纲：革螨股：巨螯螨科）

在所查到的献中,饰样小全盾螨Holostaspella ornata(Berlese)．1904仅记述雌螨(Krantz,1967;BpereTosa．1977),今根据白城标本描述其雄螨。     

革赛螨属一新种（蜱螨亚纲：革螨股：胭螨科）

记述革赛螨属1新种：敦化革赛螨Gamasellus dunhuaensis,sp．nov．,模式标本保存在吉林省白城市全国鼠疫布氏菌病防治基地。     

Quadruplexes of human telomere DNA analogs designed to contain G:A:G:A,G:G:A:A,and A:A:A:A tetrads

Replacement of two to four guanines by adenines in the human telomere DNA repeat dG₃(TTAG₃)₃ did not hinder the formation of quadruplexes if the substitutions took place in the terminal tetrad bridged by the diagonal loop of the intramolecular antiparallel three‐tetrad scaffold, as proved by CD and PAGE in both Na⁺ and K⁺ solutions. Thermodynamic data showed that, in Na⁺ solution, the dG₃(TTAG₃)₃ quadruplex was destabilized, the least by the two G:A:G:A tetrads, the most by the G:G:A:A tetrad in which the adenosines replaced syn‐guanosines. In physiological K⁺ solution, the highest destabilization was caused by the 4A tetrad. In K⁺, only the unmodified dG₃(TTAG₃)₃ quadruplex rearranged into a K⁺‐dependent quadruplex form, none of the multiple adenine‐modified structures did so. This may imply biological consequences for nonrepaired A‐for‐G mutations. © 2010 Wiley Periodicals, Inc. Biopolymers 93: 880–886, 2010.