中国野生山梨叶片形态及光合特性

以异位保存在国家梨资源圃的48份我国原产山梨品种和2份秋子梨地方品种为材料,比较了野生山梨与地方秋子梨品种间差异,研究了我国野生山梨的光合特征以及光合特性相关指标间的关系,建立了山梨光合及瞬时水分利用特征的线性回归方程.结果表明:地方品种叶片形态特征指标、叶绿素含量、光合特征指标都显著低于野生品种平均值,且低于大部分野生品种的测定值;山梨叶片的比叶面积、叶干物质含量、胞间CO2浓度的变异系数较低,其他8项指标变异系数为0.12～0.41,表现出较高的多样性水平,可见我国野生山梨资源光合特征差异明显;光合特性指标与叶绿素组成(Chl a/b)、叶干物质含量呈显著相关;光合速率与胞间CO2浓度、蒸腾速率、气孔导度呈显著的指数曲线关系,山梨光合速率主要受气孔限制的影响.‘锦州山梨’具有高光合特性,可作为山梨资源光合特征研究利用的良好材料.     

大亮子河国家森林公园野生浆果资源产业化开发利用前景分析

为了摸清大亮子河具有开发利用价值的野生浆果资源的种类、分布、用途及蕴藏量,我们于2005—2007年6—9月,通过线路调查、样地调查和访问调查的方法,对该地区野生浆果资源进行了研究。结果表明该地区有开发利用价值的野生浆果资源达20种,隶属19属14科。并对该地区野生浆果资源的产业化开发利用潜力和优势进行了深入分析,认为该地区野生浆果资源开发利用具有广阔的市场前景,最后提出开发利用过程中需要注意的问题。因此,本研究可为今后大亮子河野生浆果资源的可持续开发利用和合理保护提供科学依据。     

广东省野生水果植物资源

本文报道了广东省野生水果植物资源的调查结果,计有野生水果植物２３４种,隶属于４５科９３属。文章分析了广东野生水果植物资源的特点、开发利用的现状及存在问题,对开发利用广东野生水果资源提出了一些建议。     

中国野生杜鹃资源开发利用探讨

介绍中国野生杜鹃资源的基本特征、种类及其分布,观赏特性及开发利用价值,从而提出中国野生杜鹃资源开发利用的建议。     

云南丽江的野生花卉资源及开发利用

报道了云南丽江的野生花卉资源的特点,简要介绍了丽江分布的一些著名的野生花卉,分析了丽江野生花卉的开发利用的现状、前景,并提出了开发利用的建议.     

晋南地区三叶木通野生资源的开发利用

从三叶木通的生物学特征、化学成分、利用价值以及它的开发利用现状等方面论述了它在晋南地区三叶木通野生资源急需开发的重要性;同时提出了三叶木通野生资源开发利用发展前景及可行性建议.     

云南普洱市野生果类植物资源研究

初步查明普洱市常见的野生果类植物有59种,隶属于25科37属。对部分特色野果作了叙述,并阐述了野生果类植物开发利用的现状,提出了合理开发利用野生果类植物资源的建议。     

山西珍稀野生花卉种质资源与利用

本文论述了山西珍稀野生花卉种质资源的种类、观赏特性和应用前景,并进一步分析了其生态特点和开发利用途径,为今后山西珍稀野生花卉的研究和开发利用提参考.     

北京山区野生花卉资源及开发利用

本文论述了北京山区的野生花卉资源及其利用现状 ,分析了北京山区开发野生花卉的前景 ,指出北京山区在野生花卉的开发利用中应重点加强五个方面 (野生花卉的引种驯化、确定野生花卉的发展方向、保护性的开发利用野生花卉、加强野生花卉的生化毒性研究、加强野生花卉的育种 )工作 ,使之成为北京地区的主要产业之一。     

北京地区野生草种资源及其开发利用

本文论述了北京市的野生草种资源及其利用现状,分析了北京市开发野生草种的前景,指出北京市在野生草种的开发利用中,应重点加强野生草种的引种驯化、野生草种的新品种选育,确定野生草种的发展方面,加强宣传,增强人们对野生草种的认识,保护性的开发利用野生草种等工作,使之成为北京市的主要产业之一.     

Role for dopamine in malonate-induced damage in vivo in striatum and in vitro in mesencephalic cultures

Defects in mitochondrial energy metabolism have been implicated in the pathology of several neurodegenerative disorders. In addition, the reactive metabolites generated from the metabolism and oxidation of the neurotransmitter dopamine (DA) are thought to contribute to the damage to neurons of the basal ganglia. We have previously demonstrated that infusions of the metabolic inhibitor malonate into the striata of mice or rats produce degeneration of DA nerve terminals. In the present studies, we demonstrate that an intrastriatal infusion of malonate induces a substantial increase in DA efflux in awake, behaving mice as measured by in vivo microdialysis. Furthermore, pretreatment of mice with tetrabenazine (TBZ) or the TBZ analogue Ro 4-1284 (Ro-4), compounds that reversibly inhibit the vesicular storage of DA, attenuates the malonate-induced DA efflux as well as the damage to DA nerve terminals. Consistent with these findings, the damage to both DA and GABA neurons in mesencephalic cultures by malonate exposure was attenuated by pretreatment with TBZ or Ro-4. Treatment with these compounds did not affect the formation of free radicals or the inhibition of oxidative phosphorylation resulting from malonate exposure alone. Our data suggest that DA plays an important role in the neurotoxicity produced by malonate. These findings provide direct evidence that inhibition of succinate dehydrogenase causes an increase in extracellular DA levels and indicate that bioenergetic defects may contribute to the pathogenesis of chronic neurodegenerative diseases through a mechanism involving DA.     

Scurvy in capybaras bred in captivity in Argentine

In order to determine if the absence of vitamin C in the diet of capybaras (Hydrochoerus hydrochaeris) causes scurvy, a group of seven young individuals were fed food pellets without ascorbic acid, while another group of eight individuals received the same food with 1 g of ascorbic acid per animal per day. Animals in the first group developed signs of scurvy-like gingivitis, breaking of the incisors and death of one animal. Clinical signs appeared between 25 and 104 days from the beginning of the trial in all individuals. Growth rates of individuals deprived of vitamin C was considerably less than those observed in the control group. Deficiency of ascorbic acid had a severe effect on reproduction of another population of captive capybaras. We found that the decrease in ascorbic acid content in the diet affected pregnancy, especially during the first stages. The results obtained suggest that it is necessary to supply a suitable quantity of vitamin C in the diet of this species in captivity.     

Changes in lactate dehydrogenase activity in chicken tissues in hyperthyreosis in ontogenesis

The lactate dehydrogenase activity in reactions of lactate oxidation and synthesis was studied in subfractions of the chicken brain, heart and liver at the embryonal, early postembryonal and adult stages of development after thyroxine administration. It has been shown that during embryogenesis thyroxine predominantly enhanced the rate of lactate oxidation in the mitochondrial tissues. A marked increase in the lactate synthesis was found in cytoplasm of the adult chicken tissues. Specificity of enzyme activity alterations was detected in the chicken brain during ontogenesis after thyroxine administration.     

SSTR5 ablation in islet results in alterations in glucose homeostasis in mice

Somatostatin (SST) peptide is a potent inhibitor of insulin secretion and its effect is mediated via somatostatin receptor 5 (SSTR5) in the endocrine pancreas. To investigate the consequences of gene ablation of SSTR5 in the mouse pancreas, we have generated a mouse model in which the SSTR5 gene was specifically knocked down in the pancreatic beta cells (betaSSTR5Kd) using the Cre-lox system. Immunohistochemistry analysis showed that SSTR5 gene expression was absent in beta cells at three months of age. At the time of gene ablation, betaSSTR5Kd mice demonstrated glucose intolerance with lack of insulin response and significantly reduced serum insulin levels. Insulin tolerance test demonstrated a significant increase of insulin clearance in vivo at the same age. In vitro studies demonstrated an absence of response to SST-28 stimulation in the betaSSTR5Kd mouse islet, which was associated with a significantly reduced SST expression level in betaSSTR5Kd mice pancreata. In addition, betaSSTR5Kd mice had significantly reduced serum glucose levels and increased serum insulin levels at 12 months of age. Glucose tolerance test at an older age also indicated a persistently higher insulin level in betaSSTR5Kd mice. Further studies of betaSSTR5Kd mice had revealed elevated serum C-peptide levels at both 3 and 12 months of age, suggesting that these mice are capable of producing and releasing insulin to the periphery. These results support the hypothesis that SSTR5 plays a pivotal role in the regulation of insulin secretion in the mouse pancreas.