Increased LRRK2 kinase activity alters neuronal autophagy by disrupting the axonal transport of autophagosomes

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A Ca2+-Dependent Switch Activates Axonal Casein Kinase 2α Translation and Drives G3BP1 Granule Disassembly for Axon Regeneration

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Lipid-mediated motor-adaptor sequestration impairs axonal lysosome delivery leading to autophagic stress and dystrophy in Niemann-Pick type C

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Local translation provides the asymmetric distribution of CaMKII required for associative memory formation

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Overlapping transcriptional programs promote survival and axonal regeneration of injured retinal ganglion cells

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γ-Protocadherins control synapse formation and peripheral branching of touch sensory neurons

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Small Molecule SARM1 Inhibitors Recapitulate the SARM1−/− Phenotype and Allow Recovery of a Metastable Pool of Axons Fated to Degenerate

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Neurexin and frizzled intercept axonal transport at microtubule minus ends to control synapse formation

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Thalamic input to motor cortex facilitates goal-directed action initiation

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Regulatory imbalance between LRRK2 kinase,PPM1H phosphatase,and ARF6 GTPase disrupts the axonal transport of autophagosomes

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mRNA transport,translation, and decay in adult mammalian central nervous system axons

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Sodium channel slow inactivation normalizes firing in axons with uneven conductance distributions

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Restoring Cellular Energetics Promotes Axonal Regeneration and Functional Recovery after Spinal Cord Injury

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Regional,Layer, and Cell-Type-Specific Connectivity of the Mouse Default Mode Network

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Lateral axonal modulation is required for stimulus-specific olfactory conditioning in Drosophila

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Clathrin packets move in slow axonal transport and deliver functional payloads to synapses

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The Zonal Organization of Odorant Receptor Gene Choice in the Main Olfactory Epithelium of the Mouse

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Reprogramming an energetic AKT-PAK5 axis boosts axon energy supply and facilitates neuron survival and regeneration after injury and ischemia

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Developmental defects in Huntington’s disease show that axonal growth and microtubule reorganization require NUMA1

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A diabetic milieu increases ACE2 expression and cellular susceptibility to SARS-CoV-2 infections in human kidney organoids and patient cells

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