公立医院固定资产非效率投资内涵及度量模型研究

借鉴企业非效率投资内涵界定思路，基于公立医院新医改后的财务目标定位，采用经济学成本效益分析法对公立医院非效率投资内涵进行了界定，从而确定公立医院非效率度量方法；基于资源依赖理论，构建了公立医院非效率投资的度量理论模型，并确定了模型主要变量的选取。     

自由现金流量与公立医院过度投资关系研究 ——以B地区公立医院为例

基于自由现金流量假说理论,结合公立医院所面临的融资、盈余分配制度、医疗费用的支付方式以及公立医院面临的市场风险等,提出自由现金流量对公立医院过度投资影响的理论假设;然后,以固定资产过度投资为因变量,自由现金流量以及医院等级等为自变量,并控制医院的规模、现金存量以及上期资本支出因素,构建多元回归模型,以B地区公立医院的实际数据对研究假设进行实证检验。研究结果表明：自由现金流量是公立医院过度投资的重要诱因之一,且三级医院比二级医院更容易发生自由现金流量过度投资。     

负债融资对公立医院固定资产过度投资影响实证研究 ——以B地区公立医院为例

目的 实证分析公立医院负债融资对固定资产过度投资的影响。方法 结合公立医院所面临的制度背景,提出理论假设,并构建多元回归实证模型,以B地区公立医院的数据进行实证检验。结果 银行借款和应付账款对过度投资有显著性影响,而预付账款没有影响。结论 银行借款融资不能抑制公立医院的过度投资行为,反而是公立医院的过度投资行为重要诱因,尤其是对于大型三级公立医院来说,银行借款不仅直接为其过度投资提供资金来源,而且银行借款的杠杆收益还增加医院的自由现金流量,引发自由现金流量过度投资;应付账款能够有效抑制公立医院的过度投资行为;研究结果提示政府应当从打破公立医院垄断地位和加强公立医院商业银行借款监管两方面着手为债务治理效应创造良好环境。     

公立医院现金持有量水平评价分析

在借鉴公司现有持有相关研究的基础上,结合我国公立医院实际情况,构建实证模型,以B地区171家二级医院和三级医院2006—2009年数据验证。实证结果表明：公立医院并不像感性认识的那样不重视财务管理,就现金管理来说,医院积极进行现金管理,对其现金持有量进行动态调整,以实现目标现金持有额。以现金持有量满足日常运营的时间判断,公立医院现金持有水平是合理的。     

以政策性亏损补偿为核心的公立医院财政投入测算模型研究

收集武汉市2007—2010年二、三级综合公立医院运行的财务数据,构建公立医院财政投入测算理论模型,预测2012年武汉市二、三级综合医院财政投入金额。结果表明,以现阶段的财政投入水平为参考,公立医院可以维持其正常运行。     

我国综合性公立医院价值观倾向的特征研究

通过对我国东、中、西3省（直辖市）9个地区的78所综合性公立医院的13组价值观的倾向性调查`，试图了解目前我国综合性公立医院的价值观面貌的特征。研究显示，我国综合性公立医院价值观倾向有较大趋同性。不同战略定位类型医院分析显示，B类医院价值观倾向普遍趋向弱势，值得关注。     

公立医院职能科室绩效考核模式分析和应用

公立医院职能科室绩效考核由于缺乏同质性、度量性和激励性,成为医院绩效管理中的难点。通过对我国目前公立医院职能科室各种绩效考核模式进行分析,结合无锡市人民医院在绩效考核中相关模式的探索应用,提出改进和完善医院职能科室绩效考核管理方法。     

我国公立医院动态竞争力的构建及其探索

面临越来越复杂、多变和不可预测的竞争环境和日趋激烈的竞争压力，构建动态竞争力是公立医院发展的迫切需要。在医院动态竞争力内涵与特征的基础上，分析了公立医院动态竞争力构成要素，提出了医院动态竞争力构建的策略路径，并结合郑州大学第一附属医院构建动态竞争力的实践进行了案例研究。     

公立医院外部绩效评价的执行路径研究

目的 探索公立医院外部绩效评价在公立医院的有效执行路径。方法 访谈公立医院管理人员,调研不同执行情况的北京市属公立医院进行案例分析;构建公立医院外部绩效评价政策执行的分析框架;对公立医院外部绩效评价政策资料和研究文献进行分析。结果 构建公立医院外部绩效评价政策在公立医院的四种执行模式,归纳外部绩效评价执行的院科两级影响因素。结论 政府主管部门、公立医院、部门科室三层级的绩效评价融合是推进公立医院外部绩效评价政策在公立医院内有效执行的路径选择。     

公立医院法律顾问工作模式探析

目前公立医院面临的法律风险愈加复杂化、多元化，法律风险防范模式落后，急需调整转变。文章提出以构建总法律顾问制度为核心的公立医院法律顾问工作模式转变的路径选择，从而增强公立医院法律风险的可控性和可预防性，为公立医院科学化、规范化和法治化发展提供法律技术支撑。     

Recent advances in the study of Kaposi's sarcoma-associated herpesvirus replication and pathogenesis

It has now been over twenty years since a novel herpesviral genome was identified in Kaposi's sarcoma biopsies. Since then, the cumulative research effort by molecular biologists, virologists, clinicians, and epidemiologists alike has led to the extensive characterization of this tumor virus, Kaposi's sarcoma-associated herpesvirus(KSHV; also known as human herpesvirus 8(HHV-8)), and its associated diseases. Here we review the current knowledge of KSHV biology and pathogenesis, with a particular emphasis on new and exciting advances in the field of epigenetics. We also discuss the development and practicality of various cell culture and animal model systems to study KSHV replication and pathogenesis.     

The structure, reproduction and taxonomy of Vidalia and Osmundaria (Rhodophyta, Rhodomelaceae)

Comprises species occurring mostly in subtidal habitats in tropical, subtropical and warm-temperate areas of the world. An analysis of the type species, V. spiralis (Sonder) Lamouroux ex J. Agardh, a species from Australia, establishes basic characters for distinguishing species in the genus. These characters are (1) branching patterns of thalli, (2) flat blades that may be spiralled on their axis, (3) width of the blade, (4) primary or secondary derivation of sterile and fertile branchlets and (5) position of sterile and fertile branchlets on the thalli. Application of the latter two characters provides an important basic method for separation of species into three major groups. Osmundaria , a genus known only in southern Australia, was studied in relation to Vidalia , and its separation from the Vidalia assemblage is not accepted. Species of Vidalia therefore are transferred to the older genus name, Osmundaria. Two new species, Osmundaria papenfussii and Osmundaria oliveae are described from Natal. Confusion in the usage of the epithet, Vidalia fimbriala Brown ex Turner has been clarified, and Vidalia gregaria Falkenberg, described as an epiphyte on Osmundaria pro/ifera Lamouroux, is revealed to be young branches of the host, Osmundaria prolifera.     

New or rare chromosome counts in Artemisia L. (Asteraceae, Anthemideae) and related genera from Kazakhstan

Fifteen chromosome counts of six Artemisia taxa and one species of each of the genera Brachanthemum, Hippolytia, Kaschgaria, Lepidolopsis and Turaniphytum are reported from Kazakhstan. Three of them are new reports, two are not consistent with previous counts and the remainder are confirmations of very scarce (one to four) earlier records. All the populations studied have the same basic chromosome number, x = 9, with ploidy levels ranging from 2x to 6x. Some correlations between ploidy level, morphological characters and distribution are noted.     

肝癌中HBV和HCV基因和抗原的分布及意义

采用原位分子杂交方法检测HCV RNA及HBV X基因;采用免疫组织化学方法研究HCV核心抗原,非结构区C33c抗原及HBxAg在肝细胞肝癌中的定位及分布.结果表明(1)HCV RNA、HBV X基因在肝细胞肝癌组织检出率分别为40%(55/136)和82%(112/136).HCV RNA定位于癌细胞的胞浆内,阳性细胞呈散在、灶状及弥漫分布三种形式;HBV X基因在肝癌细胞中的分布呈胞浆型、核型及核浆型,阳性细胞也呈上述三种分布形式;(2)HCV C33c抗原、核心抗原在肝细胞肝癌中的阳性率为81%(133/164)及86%(141/164).C33c抗原定位于癌细胞及肝细胞的胞浆内;核心抗原既定位于癌细胞核中,又可定位于胞浆中.C33c抗原阳性细胞以灶状分布为主;而核心抗原阳性细     

Selection with two alleles and complete dominance

For a plant selection model with frequency-independent viabilities, fertilities and selfing rates, it is shown that apart from global fixation, for certain parameter combinations a protected polymorphism and facultative fixation (either allele may become fixed according to initial frequencies) may both occur. Facultative fixation requires different selling rates for the dominant and recessive type. Protection of the polymorphism requires resource allocation for male and female function. In this connection the problem of purely genetically caused population extinction is discussed.
For general frequency dependence and regular segregation, the chances for establishment of a completely recessive gene are compared to those of a completely dominant gene. It is proven that the process of establishment of the recessive gene, despite a fitness advantage, may be considerably endangered by drift effects if random mating prevails. The recessive gene may reach the same effectivity in establishment as a dominant gene, only if the recessive homozygote mates exclusively with its own type during the period of establishment.