共查询到20条相似文献,搜索用时 15 毫秒
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C. H. CADMAN 《The Annals of applied biology》1951,38(4):801-811
A selection of Norfolk Giant raspberry is infected with a virus transmissible by Amphorophora rubi Kalt. after short feeding periods on infected plants and persisting for at least 18 1/2 hr. in the aphid. This virus is identified with one which is carried without symptoms by Norfolk Giant and Baumforth's Seedling B, and causes necrosis on Rubus henryi and mosaic symptoms on R. saxatilis , American black raspberry R. occidentalis (var. Cumberland) and the red raspberry varieties Chartham, Mailing Landmark and St Walfried. The virus is present in some commercial stocks of Baumforth's Seedling B, Burnetholm Seedling, and the Mailing varieties Enterprise, Notable and Promise. The name raspberry leaf mottle is proposed. 相似文献
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C. H. CADMAN 《The Annals of applied biology》1952,39(1):61-68
Three diseases characterized by vein chlorosis of varying grades of severity are shown to be graft transmissible to a wide range of raspberry varieties. The diseases are thought to be caused by related strains of a virus transmissible by Doralis (Aphis) idaei V. d. G. and rarely, if at all, by Amphorophora rubi Kalt. It is proposed to refer to the diseases and viruses respectively as mild, moderate and severe vein chlorosis. 相似文献
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From the time of first feeding on plants infected with strawberry virus 3, 10–19 days elapsed before Capitophorus fragariae became infective, a longer 'latent period' than any previously recorded for an aphid-transmitted virus. The time taken for aphids to develop infectivity after leaving infected plants decreased with increasing duration of the infection feed. Aphids which had fed for 16 days on an infected plant caused infection in the first day of test feeding. 相似文献
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INVESTIGATION OF CHRYSANTHEMUM VIRUSES II. VIRUS B (MILD MOSAIC) AND CHRYSANTHEMUM LATENT VIRUS 总被引:2,自引:0,他引:2
M. HOLLINGS 《The Annals of applied biology》1957,45(4):589-602
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W. Ritchie Russell 《BMJ (Clinical research ed.)》1961,2(5246):221-222
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Cacao virus iA, the most important and prevalent of the viruses that attack cacao in the Gold Coast, occurs in strains that differ widely in their virulence towards cacao. Outbreaks usually contain trees infected with different strains and individual trees are often infected simultaneously with more than one strain; this can be demonstrated by coppicing the trees, and by inoculating sets of test plants with grafts from different parts of one tree. Neither mild nor virulent strains seemed to be consistently dominant in roots or in other parts of cacao trees.
Cacao plants infected with mild strains were nearly always protected against the effects of infection by virulent strains; however, virulent strains entered hosts already infected with mild strains, but usually without causing any symptoms unless the plants were coppiced. The severe symptoms that developed on new growth from such coppiced plants were seldom repeated in later growth. Mealybugs transmitted the virulent strains from leaves with symptoms characteristic of infection by the latter, but not from leaves free from such symptoms. These results suggest that the multiplication of a virulent strain is impeded in plants infected with a mild strain.
In the field, infection with a mild strain protected mature trees against the effects of virulent strains spread by mealybugs. During 3 years in which 273 out of 387 previously uninfected trees became severely diseased, only 35 out of 416 infected with themildstrain developed symptoms of infectionwiththevirulent strain. Five years after infection with the mild strain, trees were yielding I pod per tree more than in the year they were infected, whereas the decrease on trees infected with the virulent strain was 16 pods per tree. Some limitations in the practical application of protection by mild strains, and objections to its use as a control measure, are discussed. 相似文献
Cacao plants infected with mild strains were nearly always protected against the effects of infection by virulent strains; however, virulent strains entered hosts already infected with mild strains, but usually without causing any symptoms unless the plants were coppiced. The severe symptoms that developed on new growth from such coppiced plants were seldom repeated in later growth. Mealybugs transmitted the virulent strains from leaves with symptoms characteristic of infection by the latter, but not from leaves free from such symptoms. These results suggest that the multiplication of a virulent strain is impeded in plants infected with a mild strain.
In the field, infection with a mild strain protected mature trees against the effects of virulent strains spread by mealybugs. During 3 years in which 273 out of 387 previously uninfected trees became severely diseased, only 35 out of 416 infected with themildstrain developed symptoms of infectionwiththevirulent strain. Five years after infection with the mild strain, trees were yielding I pod per tree more than in the year they were infected, whereas the decrease on trees infected with the virulent strain was 16 pods per tree. Some limitations in the practical application of protection by mild strains, and objections to its use as a control measure, are discussed. 相似文献
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Jaques Bourdillon 《The Journal of general physiology》1941,25(2):263-273
Analytical diffusion has been applied to a study of influenza A virus in mouse lung, influenza A virus in the extra-embryonic fluids of the chick, and mouse encephalomyelitis virus in mouse brain. The results from influenza in mouse lung suggested that about 99 per cent of the infectivity was present in particles 200 mµ in diameter, and 1 per cent in particles 6 mµ in diameter. The results from influenza in extra-embryonic fluids indicated that the preparation was inhomogeneous and that the smallest virus units were about 6 mµ in diameter. The results from mouse encephalomyelitis virus indicated that the preparation was also inhomogeneous, with 10 per cent of the infectivity in particles about 15 mµ in diameter. It has been suggested that in virus preparations normal colloidal particles can act as carriers of much smaller virus units. 相似文献
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M. C. Williams 《Ibis》1957,99(2):303-306
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牛病毒性腹泻病毒(BVDV)对牛免疫缺陷病毒(BIV)的激活作用 总被引:6,自引:0,他引:6
通过合胞体分析和反转录酶活力测定,首次证明牛病毒性腹泻病毒能激活牛免疫缺陷病毒的复制与表达,并进一步通过转染实验和凝胶电泳漂移分析证明,当BIV LTR的NF-k B区缺失时,BVDV则不能实现其激活作用,BVDV直接或间接诱导牛NF-kB因子作用于BIV LTR的NF-kB区实现其激活作用。 相似文献
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