应用脉冲多普勒超声心动图观察体外反搏（吸氧）对左室舒张功能的影响

脉冲多普勒超声心动图仪了解体外反搏(吸氧)对左室舒张功能的作用.方法全部受试者采用HP Sonos 100多功能超声心动图仪做常规脉冲多普勒检查,同步记录心电图,观察患者体外反搏(吸氧)治疗前后左室舒张功能的多个参数.体外反搏(吸氧)治疗后VE/VAEi/Ai的比值分别提高了22%,35%(P＜0.001).体外反搏(吸氧)治疗对左室舒张功能改善确有一定作用.     

高强度间歇训练心脏康复对冠心病患者PCI术后心脏功能及应激因子的影响

摘要 目的：探讨高强度间歇训练心脏康复对冠心病患者经皮冠状动脉介入（PCI）术后心脏功能及应激因子的影响。方法：选择我院于2017年3月至2019年3月行PCI术冠心病患者86例,采用随机数字表法随机分为观察组43例与对照组43例。观察组患者采用高强度间歇训练心脏康复,对照组患者采用常规心脏康复。两组疗程均为12周。比较两组康复前后心脏功能、运动耐力、应激因子及生活质量影响。结果：观察组康复后左室射血分数（LVEF）和心输出量高于对照组（P<0.05）。观察组康复后峰值功率（PP）、运动持续时间（ED）和峰值摄氧量（VQ2peak）高于对照组（P<0.05）。观察组康复后C反应蛋白（CRP）、肿瘤坏死因子-α（TNF-α）和白介素-6（IL-6）水平低于对照组（P<0.05）。观察组康复后心绞痛稳定程度、疾病主观感受、躯体活动受限程度、心绞痛发作频率和治疗满意程度评分高于对照组（P<0.05）。结论：高强度间歇训练心脏康复可改善冠心病PCI术后心脏功能,减轻应激反应,改善患者运动耐力及生活质量。     

训练专一性对无氧阈测定的影响

本文对以上肢训练为主的15名优秀皮划艇运动员和以下肢训练为主的14名优秀中长跑运动员在两种常规负荷方式下的无氧阈及最大吸氧量进行了测定分析,以探讨训练专一性对无氧阈测定的影响。采用踏车式功量计和活动平板方式逐级递增负荷,通过与Apple Ⅱ_E辅助联机的Jaeger EOS自动分析系统,以通气和气体交换指标的变化,无创性地测定无氧阈和最大吸氧量。结果表明,训练专一性会影响无氧阈测定结果。欲测得最高的无氧阈值,实验室测试手段就应当尽量模拟训练时的运动形式。此外,能敏感地反映出运动训练专一性适应的是无氧阈时的吸氧量绝对值(1/min),而不是无氧阈的相对值(％Vo_2max)。     

强化间歇训练对慢性心衰患者心肺耐力及自主神经功能的影响

目的：探讨强化间歇训练对慢性心衰患者心肺耐力及自主神经功能的影响。方法：34名稳定期慢性心衰患者随机分成试验组和对照组,每组各17名,两组均采用常规药物治疗,其中试验组同时结合间歇运动训练治疗,训练周期12周,前2周为适应性训练,采用功率自行车和30%~50% HRmax强度进行训练,每周训练3次,每次运动20~30 min;后10周采用强化间歇训练,采用功率自行车和运动靶心率=80%~90% HRmax强度训练,训练时间30~40 min,每周训练3次;采用6 min步行距离和心率变异性指标进行效果评价。结果：与试验前比较,12周治疗后,对照组和试验组6 min步行距离均增加,差异具有统计学意义（P<0.01）,反映自主神经功能的时域指标和频域指标均出现增高,差异具有统计学意义（P<0.01）;试验后,试验组6 min步行距离、时域指标和频域指标改变幅度大于对照组,差异具有统计学意义（P<0.01）。结论：强化间歇训练可以作为慢性心衰患者心肺耐力提高和自主神经功能改善的康复手段,比单一的药物治疗效果更好。     

运动训练和体位对安静负荷及恢复期心功能的影响

运动及恢复过程中心功能改变及运动训练对心功能影响的研究虽有不少报道,但关于多级小档距递增负荷中的心功能变化及不同体位对此变化的影响则报道甚少。本实验用无创法测定了不同体能的人在坐位和卧位下完成相同亚极量递增负荷过程中及恢复期的心功能指标,旨在进一步探讨运动负荷中及恢复期的心功能特点及运动训练和体位改变对它的影响。     

补服支链氨基酸对有氧运动耐力的影响

为了研究补服支链氨基酸(branched-chain amino acid,BCAA)对有氧运动耐力的影响,给白鼠补服BCAA,检测其对白鼠的运动能力和血清游离氨基酸代谢的影响;另选择健康男性作为受试者,于测试前30min分别口服氨基酸补剂及安慰剂,采取自身对照性分析法对比12min跑成绩及最大吸氧量的变化。结果表明,白鼠补充BCAA后,有氧运动的衰竭推迟出现,血清中BCAA的含量显著提高;人体数据研究表明,补服BCAA后,12min跑成绩和最大吸氧量均显著提高。表明补服BCAA能有效提高人体的有氧运动耐力。     

间歇运动对心梗大鼠梗死周边区单个心肌细胞钙瞬变和收缩功能的影响

目的:探讨间歇运动对心梗大鼠缺血心肌细胞钙瞬变和收缩功能改变的影响。方法:3月龄SD雄性大鼠24只,适应性喂养1周后随机分为假手术组(S)、心梗组(MI)、心梗+运动组(ME),每组8只。MI组结扎左冠状动脉前降支制备心梗模型;S组只穿线不结扎;ME组术后一周开始间歇训练,运动方式为1周适应性运动(10 m/min×30 min/d),然后先以10 m/min×10 min,再以15 m/min×6 min和25 m/min×4 min依次交替运功,60 min/d,每周5 d连续8周。训练结束后次日,腹腔麻醉并分离心肌细胞。采用单细胞可视化动缘探测系统(IonOptix)测定[Ca^2+]i amplitude、[Ca^2]+i荧光比率(Ratio)、Departure velocity、TTP、TTP50%、TTB50%、Return velocity以及Ratio amplitude等钙瞬变指标和±dl/dtmax、SL、PTA、SL shortening%等心肌细胞收缩指标。结果:与S组相比,MI组[Ca^2+]i amplitude、[Ca^2+]i Ratio amplitude,Departure velocity和Return velocity均显著下降(P<0.01),TTB50%、TTP和TTP50%均显著增加(P<0.01),心肌细胞肌节SL Shortening%、PTA、±dl/dtmax均显著减少(P<0.01);与MI组相比,ME组Ratio amplitude、[Ca^2+]i amplitude、Return velocity和Departure velocity均显著提高(P<0.01),TTB50%、TTP和TTP50%均显著缩短(P<0.01,P<0.05),心肌细胞肌节SL Shortening%、PTA、±dl/dtmax均显著提高(P<0.01,P<0.05)。结论:间歇运动可同步改善MI大鼠梗死周边区心肌活细胞的钙瞬变异常和心肌细胞的收缩功能。     

鼎突多刺蚁热适应及运动行为的热依赖性

将鼎突多刺蚁(Polyrhachis vicina)分别置于15、20、25和30℃的恒温恒湿培养箱内,对其热适应及运动行为的热依赖性进行了研究。驯化2周后,采用温度梯度仪测量其热适应参数,并选用停顿频率(PF)、疾跑速度(SS)和最大持续运动距离(MDCCL)来衡量其运动能力。结果表明,驯化温度对鼎突多刺蚁的热适应和运动行为有极显著影响(P<0.01)。最适温度(PT)、临界低温(CLT)、临界高温(CHT)随驯化温度(AT)的升高而增大,最终适温为30.54℃,临界低温不低于3℃,临界高温不高于45℃;经过高温驯化的鼎突多刺蚁的运动能力显著大于经过低温驯化的个体运动能力,驯化温度与疾跑速度、最大持续运动距离呈显著的正相关,而与停顿频率呈极显著的负相关。     

持续与间歇重复跑台运动对大鼠空间学习记忆能力及海马BDNF基因表达的影响

目的：探讨中等强度持续与间歇重复跑台运动6周对大鼠学习记忆能力及海马内脑源性神经营养因子（BDNF）基因表达的影响。方法：将24只雄性SD大鼠随机分为3组（n=8）：空白对照（SC）组、持续训练（CT）组和间歇训练（IT）组。持续训练组以24 m/min的跑速持续运动30 min,训练频度每日1次;IT组与CT组运动强度和运动总时间相同,但IT组将30 min运动时间分为2个训练单位,每个单位15 min,两个训练单位间隔1 h,共训练6周。训练结束后,采用Morris水迷宫实验测试空间定位能力和探索能力。最后一次运动24 h后开颅取海马,QT-PCR法检测BDNF mRNA的表达水平。结果：①定位航行实验的结果表明,与SC组相比,IT组和CT组大鼠的逃避潜伏期显著缩短（P<0.01）,周围路程与周围时间均显著下降（P<0.05,P<0.01）,IT组与CT组之间无统计学差异。②空间探索实验的结果表明,与SC组相比,CT组和IT组站台中心路程均显著增加（P<0.05）,周围时间均显著减少（P<0.05,P<0.01）,但CT组和IT组组间相比无统计学差异;与SC组和CT组相比,IT组大鼠的潜伏期、站台周围时间均显著减少（P<0.05）。③QT-PCR结果显示,与SC组与CT组相比,IT组BDNF mRNA表达水平显著增高。结论：6周间歇重复跑台训练明显改善大鼠的空间学习记忆能力,这可能与这种类型运动更有利于促进海马BDNF基因的高表达有关。     

缩唇腹式呼吸训练联合弹力带抗阻运动对慢性心力衰竭患者运动耐力、心肺功能及生活质量的影响

摘要 目的：观察缩唇腹式呼吸训练对联合弹力带抗阻运动对慢性心力衰竭（CHF）患者运动耐力、心肺功能及生活质量的影响。方法：选取2020年4月~2021年7月期间我院收治的CHF患者83例。按照双色球法将患者分为对照组（n=41）和观察组（n=42）,对照组接受弹力带抗阻运动,观察组接受缩唇腹式呼吸训练联合弹力带抗阻运动。观察两组运动耐力、心肺功能、生活质量及1年内再住院率和1年内死亡率情况。结果：两组干预4周后躯体领域评分、情绪领域评分、其他领域评分和总分均下降,且观察组低于对照组（P<0.05）。两组干预4周后用力肺活量（FVC）、第1秒用力呼气容积（FEV₁）、最大自主分钟通气量（MVV）、左室射血分数（LVEF）升高,且观察组高于对照组（P<0.05）,而左室舒张末期内径（LVEDD）、左室收缩末期内径（LVESD）下降,且观察组低于对照组（P<0.05）。两组干预4周后6 min步行距离试验（6MWT）、峰值摄氧量（VO_2peak ）及无氧阈值（AT）升高,且观察组高于对照组（P<0.05）。观察组的1年内再住院率、1年内死亡率均低于对照组（P<0.05）。结论：弹力带抗阻运动联合缩唇腹式呼吸训练可促进CHF患者心肺功能改善,提高运动耐力,促进生活质量提升,同时还可降低1年内再住院率、1年内死亡率,疗效较好。     

Cardiovascular adaptations to exercise training in the elderly

Maximal O2 uptake (VO2max) and left ventricular function decrease with age. Endurance exercise training of sufficient intensity, frequency, and duration increases VO2max in the elderly. The mechanisms underlying the increased VO2max in the elderly are enhanced O2 extraction of trained muscle during maximal exercise leading to a wider arteriovenous O2 difference, and higher cardiac output in the trained state. However, increased cardiac output during true maximal exercise has not been documented in elderly subjects. Endurance exercise training results in a lower heart rate and rate pressure product during submaximal exercise at a given intensity. However, no improvement in left ventricular function has been reported in the elderly after exercise training. Highly trained master athletes exhibit proportional increases in the left ventricular end-diastolic dimension and wall thickness suggestive of volume-overload hypertrophy compared with age-matched sedentary controls. The magnitude of left ventricular enlargement is similar to that in young athletes. The failure of exercise training to alter the age-related deterioration of left ventricular function in the elderly may reflect an insufficient training stimulus rather than the inability of the heart to adapt to training in elderly subjects.     

The effects of nightly normobaric hypoxia and high intensity training under intermittent normobaric hypoxia on running economy and hemoglobin mass.

We investigated the effects of nightly intermittent exposure to hypoxia and of training during intermittent hypoxia on both erythropoiesis and running economy (RE), which is indicated by the oxygen cost during running at submaximal speeds. Twenty-five college long- and middle- distance runners [maximal oxygen uptake (Vo(2max)) 60.3 +/- 4.7 ml x kg(-1) x min(-1)] were randomly assigned to one of three groups: hypoxic residential group (HypR, 11 h/night at 3,000 m simulated altitude), hypoxic training group (HypT), or control group (Con), for an intervention of 29 nights. All subjects trained in Tokyo (altitude of 60 m) but HypT had additional high-intensity treadmill running for 30 min at 3,000 m simulated altitude on 12 days during the night intervention. Vo(2) was measured at standing rest during four submaximal speeds (12, 14, 16, and 18 km/h) and during a maximal stage to volitional exhaustion on a treadmill. Total hemoglobin mass (THb) was measured by carbon monoxide rebreathing. There were no significant changes in Vo(2max), THb, and the time to exhaustion in all three groups after the intervention. Nevertheless, HypR showed approximately 5% improvement of RE in normoxia (P < 0.01) after the intervention, reflected by reduced Vo(2) at 18 km/h and the decreased regression slope fitted to Vo(2) measured during rest position and the four submaximal speeds (P < 0.05), whereas no significant corresponding changes were found in HypT and Con. We concluded that our dose of intermittent hypoxia (3,000 m for approximately 11 h/night for 29 nights) was insufficient to enhance erythropoiesis or Vo(2max), but improved the RE at race speed of college runners.     

Effect of central hypervolemia on cardiac performance during exercise

To investigate the effect of different levels of central blood volume on cardiac performance during exercise, M-mode echocardiography was utilized to determine left ventricular size and performance during cycling exercise in the upright posture (UP), supine posture (SP), and head-out water immersion (WI). At submaximal work loads requiring a mean O2 consumption (Vo2) of 1.2 1/min and 1.5 1/min, mean left ventricular end-diastolic and end-systolic dimensions were significantly greater (P less than 0.05) with WI than UP. In the SP during exercise, left ventricular dimensions were intermediate between UP and WI. Heart rate did not differ significantly among the three conditions at rest and at submaximal exercise up to a mean Vo2 of 1.8 1/min. However, at a mean Vo2 of 2.4 1/min, heart rate in the UP was significantly greater than WI (P less than 0.01) and the SP (P less than 0.05). Maximal Vo2 did not differ statistically in the three conditions. These data indicate that a change in central blood volume results in alterations in left ventricular end-diastolic and end-systolic dimensions during moderate levels of exercise and a change in heart rate at heavy levels of exercise.     

Dynamics of changes in the cardiovascular response to submaximal exercise during low-intensity endurance training with particular reference to the systolic time intervals

Eighteen male volunteers (aged 20-23 years), not involved in any sporting activities, were submitted to 13 weeks of training consisting of 30 min exercise [at 50%-75% maximal oxygen intake (VO2max)] on a cycle ergometer, performed 3 times a week. Every 4 weeks cardiac function was evaluated by measuring the systolic time intervals at rest and during submaximal cycle exercise. Stroke volume (SV), heart rate (HR) and blood pressure (BP) responses to submaximal exercise, VO2max and anaerobic threshold (AT) were also determined. Significant increases in VO2max, increases in AT and SV at the submaximal exercise intensities, as well as decreases in HR and BP were found after 4 weeks of training. Resting systolic time intervals were not affected by training, but during the submaximal cycle exercise the values of the pre-ejection period (PEP) and isovolumic contraction time (ICT) corresponding to HR of 100 beats.min-1 were significantly lowered after 13 weeks of training, whereas PEP, ICT and total electromechanical systole corresponding to HR of 130 beats.min-1 were significantly shortened by the 4th week. The ratios of PEP:LVET (left ventricular ejection time) and ICT:LVET during submaximal exercise were significantly lowered by training starting from the 8th week. These changes might be interpreted as evidence of the training-induced enhancement of the "contractility reserve", i.e. the ability to increase heart muscle contractility with increasing exercise intensity.     

A new non-exercise-based Vo2max prediction equation for aerobically trained men

The purposes of the present study were to (a) modify previously published Vo(2)max equations using the constant error (CE = mean difference between actual and predicted Vo(2)max) values from Malek et al. (28); (b) cross-validate the modified equations to determine their accuracy for estimating Vo(2)max in aerobically trained men; (c) derive a new non- exercise-based equation for estimating Vo(2)max in aerobically trained men if the modified equations are not found to be accurate; and (d) cross-validate the new Vo(2)max equation using the predicted residual sum of squares (PRESS) statistic and an independent sample of aerobically trained men. One hundred and fifty-two aerobically trained men (Vo(2)max mean +/- SD = 4,154 +/- 629 ml.min(-1)) performed a maximal incremental test on a cycle ergometer to determine actual Vo(2)max. An aerobically trained man was defined as someone who had participated in continuous aerobic exercise 3 or more sessions per week for a minimum of 1 hour per session for at least the past 18 months. Nine previously published Vo(2)max equations were modified for use with aerobically trained men. The predicted Vo(2)max values from the 9 modified equations were compared to actual Vo(2)max by examining the CE, standard error of estimate (SEE), validity coefficient (r), and total error (TE). Cross-validation of the modified non-exercise-based equations on a random subsample of 50 subjects resulted in a %TE > or = 13% of the mean of actual Vo(2)max. Therefore, the following non-exercise-based Vo(2)max equation was derived from a random subsample of 112 subjects: Vo(2)max (ml.min(-1)) = 27.387(weight in kg) + 26.634(height in cm) - 27.572(age in years) + 26.161(h.wk(-1) of training) + 114.904(intensity of training using the Borg 6-20 scale) + 506.752(natural log of years of training) - 4,609.791 (R = 0.82, R(2) adjusted = 0.65, and SEE = 378 ml.min(-1)). Cross-validation of this equation on the remaining sample of 40 subjects resulted in a %TE of 10%. Therefore, the non-exercise-based equation derived in the present study is recommended for estimating Vo(2)max in aerobically trained men.     

Influence of high-intensity interval training on adaptations in well-trained cyclists

The purpose of the present study was to examine the influence of 3 different high-intensity interval training regimens on the first and second ventilatory thresholds (VT(1) and VT(2)), anaerobic capacity (ANC), and plasma volume (PV) in well-trained endurance cyclists. Before and after 2 and 4 weeks of training, 38 well-trained cyclists (Vo(2)peak = 64.5 +/- 5.2 ml.kg(-1).min(-1)) performed (a) a progressive cycle test to measure Vo(2)peak, peak power output (PPO), VT(1), and VT(2); (b) a time to exhaustion test (T(max)) at their Vo(2)peak power output (P(max)); and (c) a 40-km time-trial (TT(40)). Subjects were assigned to 1 of 4 training groups (group 1: n = 8, 8 x 60% T(max) at P(max), 1:2 work-recovery ratio; group 2: n = 9, 8 x 60% T(max) at P(max), recovery at 65% maximum heart rate; group 3: n = 10, 12 x 30 seconds at 175% PPO, 4.5-minute recovery; control group: n = 11). The TT(40) performance, Vo(2)peak, VT(1), VT(2), and ANC were all significantly increased in groups 1, 2, and 3 (p < 0.05) but not in the control group. However, PV did not change in response to the 4-week training program. Changes in TT(40) performance were modestly related to the changes in Vo(2)peak, VT(1), VT(2), and ANC (r = 0.41, 0.34, 0.42, and 0.40, respectively; all p < 0.05). In conclusion, the improvements in TT(40) performance were related to significant increases in Vo(2)peak, VT(1), VT(2), and ANC but were not accompanied by significant changes in PV. Thus, peripheral adaptations rather than central adaptations are likely responsible for the improved performances witnessed in well-trained endurance athletes following various forms of high-intensity interval training programs.     

Effects of short-term training using powercranks on cardiovascular fitness and cycling efficiency

Powercranks use a specially designed clutch to promote independent pedal work by each leg during cycling. We examined the effects of 6 wk of training on cyclists using Powercranks (n=6) or normal cranks (n=6) on maximal oxygen consumption (VO2max) and anaerobic threshold (AT) during a graded exercise test (GXT), and heart rate (HR), oxygen consumption (VO2), respiratory exchange ration (RER), and gross efficiency (GE) during a 1-hour submaximal ride at a constant load. Subjects trained at 70% of VO2max for 1 h.d(-1), 3 d.wk(-1), for 6 weeks. The GXT and 1-hour submaximal ride were performed using normal cranks pretraining and posttraining. The 1-hour submaximal ride was performed at an intensity equal to approximately 69% of pretraining VO2max with VO2, RER, GE, and HR determined at 15-minute intervals during the ride. No differences were observed between or within groups for VO2max or AT during the GXT. The Powercranks group had significantly higher GE values than the normal cranks group (23.6 +/- 1.3% versus 21.3 +/- 1.7%, and 23.9 +/- 1.4% versus 21.0 +/- 1.9% at 45 and 60 min, respectively), and significantly lower HR at 30, 45, and 60 minutes and VO2 at 45 and 60 minutes during the 1-hour submaximal ride posttraining. It appears that 6 weeks of training with Powercranks induced physiological adaptations that reduced energy expenditure during a 1-hour submaximal ride.     

Pulmonary ventilation, blood gases, and blood pH after training of the arms or the legs.

In two groups of young healthy subjects who performed arm training (N = 5) and leg training (N = 5), respectively, the respiratory adaptation to submaximal exercise with trained and nontrained muscle groups was compared by measurement of the ventilatory equivalent (Ve/Vo2, pH, and blood gases (Pco2, Po2, and So2) in arterial blood and in venous blood from exercising extremities. After training Ve/Vo2 was significantly reduced during exercise with trained muscles, but unchanged during exercise with nontrained muscles. The reduction in Ve/Vo2 was closely related to a less pronounced increase in heart rate and in arterial lactate content, but showed no quantitative correlation to changes in arterial adaptations in trained muscles are mainly responsible for the reduction in Ve/Vo2. After training during exercise with trained as well as nontrained muscles a shift to the right of the blood oxygen dissociation curve occurred as extremities was lower while corresponding Po2 was higher.     

The effect of long-term aerobic exercise on maximal oxygen consumption,left ventricular function and serum lipids in elderly women

The purpose of this study was to investigate the changes of maximal oxygen consumption, left ventricular function and serum lipids after 36 weeks of aerobic exercise in elderly women without the influence of drugs. Eight elderly women were studied by M-mode and Doppler echocardiography to assess left ventricular size, mass and function. Maximal oxygen consumption (VO(2)max) was determined for each subject by administering a treadmill exercise test. The training intensity was decided by heart rate reserve. Subjects performed exercise for 40 minutes a day, 3 days a week at 50-60% of the heart rate reserve during the 36 weeks. Exercise capacity was assessed by VO(2)max with a graded exercise test of the treadmill. Weight and % body fat decreased after training. Cardiorespiratory function improved because of the increase in VO(2)max and VO(2)max normalized for body weight after training. Systolic blood pressure significantly decreased. There are no significant difference in all left ventricular's parameters (end-diastolic dimension, end-systolic dimension, end-diastolic volume, end-systolic volume, stroke volume, cardiac output, ejection fraction, fractional shortening) after 36 weeks. Exercise training did not induce left ventricular (LV) enlargement as evidence of an absence of increase in left ventricular end-diastolic volume. The total cholesterol level and triglyceride level decreased after training. High density lipoprotein-cholesterol significantly increased and low density lipoprotein-cholesterol significantly decreased, atherogenic index (AI) significantly decreased and apolipoprotein A-I increased and apolipoprotein B decreased after training. In conclusion, although there was no significant change in left ventricular function, aerobic training showed a positive influence on body composition, maximal oxygen consumption and serum lipids.     

Effect of concurrent endurance and circuit resistance training sequence on muscular strength and power development

The purpose of this study was to examine the influence of the sequence order of high-intensity endurance training and circuit training on changes in muscular strength and anaerobic power. Forty-eight physical education students (ages, 21.4 +/- 1.3 years) were assigned to 1 of 5 groups: no training controls (C, n = 9), endurance training (E, n = 10), circuit training (S, n = 9), endurance before circuit training in the same session, (E+S, n = 10), and circuit before endurance training in the same session (S+E, n = 10). Subjects performed 2 sessions per week for 12 weeks. Resistance-type circuit training targeted strength endurance (weeks 1-6) and explosive strength and power (weeks 7-12). Endurance training sessions included 5 repetitions run at the velocity associated with Vo2max (Vo2max) for a duration equal to 50% of the time to exhaustion at Vo2max; recovery was for an equal period at 60% Vo2max. Maximal strength in the half squat, strength endurance in the 1-leg half squat and hip extension, and explosive strength and power in a 5-jump test and countermovement jump were measured pre- and post-testing. No significant differences were shown following training between the S+E and E+S groups for all exercise tests. However, both S+E and E+S groups improved less than the S group in 1 repetition maximum (p < 0.01), right and left 1-leg half squat (p < 0.02), 5-jump test (p < 0.01), peak jumping force (p < 0.05), peak jumping power (p < 0.02), and peak jumping height (p < 0.05). The intrasession sequence did not influence the adaptive response of muscular strength and explosive strength and power. Circuit training alone induced strength and power improvements that were significantly greater than when resistance and endurance training were combined, irrespective of the intrasession sequencing.