四川省阳蝇属五新种（双翅目：蝇科）

记述采自中国四川西部山区阳蝇属Helina R.-D.,1980的5新种,分别命名为金阳蝇Helina aureolicolorata,sp.nov.;羽胫阳蝇Helina plumipostitibia,sp.nov.;茂汶阳蝇Helina maowenna,sp.nov.;雨阳蝇Helina hyeta,sp.nov.;类介阳蝇Helina mimintermedia,sp.nov.。模式标本存北京军事医学科学院微生物流行病研究所医学昆虫标本馆。     

中国四川阳蝇属三新种(双翅目:蝇科)

报道采自四川西部阳蝇属Helina R.D.,1830的3个新种：雅安阳蝇Helina yaanensis sp.nov.;花阳蝇He-lina floscula sp.nov.;类宽角阳蝇Helina inflatoides sp.nov.。模式标本保存于军事医学科学院微生物流行病研究所医学昆虫标本馆（北京）。     

新疆阳蝇属四新种(双翅目:蝇科)

报告采自新疆蝇科阳蝇属Helina R.-D.4新种,即眼鬃阳蝇Helina ocellijuba sp.nov.,粘叶阳蝇Helina mallocerca sp.nov.,狭跗阳蝇Helina stenotarsis sp.nov.和板刺阳蝇Helina sterniteoacaena sp.nov.。模式标本保存于中国科学院动物研究所。     

中国四川阳蝇属五新种(双翅目:蝇科)

报告采自四川西部山区的蝇科阳蝇属HelinaR.-D.五新种,即：亚喜密阳蝇Helina subevecta sp.nov,阳阳蝇Helina solata sp.nov,用公山阳蝇Helina zhougongshanna sp.nov,康定阳蝇Helina kangdingen-sis sp.nov和赘脉阳蝇Helina appendicivena sp.nov。模式标本保存于中国军事医学科学院微生物流行病研究所医学昆虫标本馆。     

中国阳蝇属国上新种（双翅目：蝇科）

报道采自黑龙江和甘两省的蝇科阳蝇属Helina R.-D.4新各上,分别命名为胡氏阳蝇Helina huae sp.nov.,小兴安阳蝇Heliona xiaoxinganna sp.nov.,舟曲阳蝇Helina zhouquensis sp.nov和圆板阳蝇Helina ampyxocerca sp.nov.。模式标本保存于沈阳师范大学昆虫研究所。     

中国阳蝇属四新种(双翅目: 蝇科)

报道采自黑龙江和甘肃两省的蝇科阳蝇属Helina R. -D. 残轮?分别命名为胡氏阳蝇Helina huae sp. nov., 小兴安阳蝇Heliona xiaoxinganna sp. nov., 舟曲阳蝇Helina zhouquensis sp. nov. 和圆板阳蝇Helina ampyxocerca sp. nov..模式标本保存于沈阳师范大学昆虫研究所.     

长白山阳蝇属三新种(双翅目:蝇科)

报道采自吉林省长白山蝇科阳蝇属Helina R.-D.3新种,即密胡阳蝇Helina densibarbata sp.nov。,吉林阳蝇Helina jilinensis sp.nov.和小毁阳蝇Helina minutideleta sp.nov.,模式标本保存于沈阳师范大学昆虫研究所。     

青海省阳蝇属四新种(双翅目,蝇科)

报告采自青海省蝇科阳蝇属Helina R—D．4新种,分别命名为高居阳蝇Helina alpigenus sp．nov．,尖尾阳蝇Helina apicicauda sp．nov．,二刺阳蝇Helina bispina sp．nov．和大毁阳蝇Helina largideleta sp．nov．。模式标本保存于中国科学院动物研究所。     

中国四川省阳蝇属五新种(双翅目:蝇科)

报道采自中国四川省阳蝇属Helina R．-D．,1830 5新种：腹刺阳蝇Helina sternitoscola,sp.nov．;华西阳蝇Helina sinoccidentala,sp．nov．;亚蜜阳蝇Helina subfica,sp．nov．;双叉阳蝇Helina didicrocerca,sp．nov．：四川阳蝇Helina sichuanica,sp．nov．。前4种模式标本保存在中国科学院上海生命科学研究院植物生理生态研究所：最后1种保存于北京军事医学科学院微生物流行病研究所医学昆虫标本馆。     

新疆阳蝇属Helina R.—D.二新种（双翅目：蝇科）

记述采自新疆蝇属Helina R.-D．2新种,托木尔阳蝇Helina tuomuerra,sp,nov。和新疆阳蝇Helina xinjiangensis,sp.nov.模式标本保存于中国科学院动物研究所。     

Recent advances in the study of Kaposi's sarcoma-associated herpesvirus replication and pathogenesis

It has now been over twenty years since a novel herpesviral genome was identified in Kaposi's sarcoma biopsies. Since then, the cumulative research effort by molecular biologists, virologists, clinicians, and epidemiologists alike has led to the extensive characterization of this tumor virus, Kaposi's sarcoma-associated herpesvirus(KSHV; also known as human herpesvirus 8(HHV-8)), and its associated diseases. Here we review the current knowledge of KSHV biology and pathogenesis, with a particular emphasis on new and exciting advances in the field of epigenetics. We also discuss the development and practicality of various cell culture and animal model systems to study KSHV replication and pathogenesis.     

The structure, reproduction and taxonomy of Vidalia and Osmundaria (Rhodophyta, Rhodomelaceae)

Comprises species occurring mostly in subtidal habitats in tropical, subtropical and warm-temperate areas of the world. An analysis of the type species, V. spiralis (Sonder) Lamouroux ex J. Agardh, a species from Australia, establishes basic characters for distinguishing species in the genus. These characters are (1) branching patterns of thalli, (2) flat blades that may be spiralled on their axis, (3) width of the blade, (4) primary or secondary derivation of sterile and fertile branchlets and (5) position of sterile and fertile branchlets on the thalli. Application of the latter two characters provides an important basic method for separation of species into three major groups. Osmundaria , a genus known only in southern Australia, was studied in relation to Vidalia , and its separation from the Vidalia assemblage is not accepted. Species of Vidalia therefore are transferred to the older genus name, Osmundaria. Two new species, Osmundaria papenfussii and Osmundaria oliveae are described from Natal. Confusion in the usage of the epithet, Vidalia fimbriala Brown ex Turner has been clarified, and Vidalia gregaria Falkenberg, described as an epiphyte on Osmundaria pro/ifera Lamouroux, is revealed to be young branches of the host, Osmundaria prolifera.     

New or rare chromosome counts in Artemisia L. (Asteraceae, Anthemideae) and related genera from Kazakhstan

Fifteen chromosome counts of six Artemisia taxa and one species of each of the genera Brachanthemum, Hippolytia, Kaschgaria, Lepidolopsis and Turaniphytum are reported from Kazakhstan. Three of them are new reports, two are not consistent with previous counts and the remainder are confirmations of very scarce (one to four) earlier records. All the populations studied have the same basic chromosome number, x = 9, with ploidy levels ranging from 2x to 6x. Some correlations between ploidy level, morphological characters and distribution are noted.     

肝癌中HBV和HCV基因和抗原的分布及意义

采用原位分子杂交方法检测HCV RNA及HBV X基因;采用免疫组织化学方法研究HCV核心抗原,非结构区C33c抗原及HBxAg在肝细胞肝癌中的定位及分布.结果表明(1)HCV RNA、HBV X基因在肝细胞肝癌组织检出率分别为40%(55/136)和82%(112/136).HCV RNA定位于癌细胞的胞浆内,阳性细胞呈散在、灶状及弥漫分布三种形式;HBV X基因在肝癌细胞中的分布呈胞浆型、核型及核浆型,阳性细胞也呈上述三种分布形式;(2)HCV C33c抗原、核心抗原在肝细胞肝癌中的阳性率为81%(133/164)及86%(141/164).C33c抗原定位于癌细胞及肝细胞的胞浆内;核心抗原既定位于癌细胞核中,又可定位于胞浆中.C33c抗原阳性细胞以灶状分布为主;而核心抗原阳性细     

Selection with two alleles and complete dominance

For a plant selection model with frequency-independent viabilities, fertilities and selfing rates, it is shown that apart from global fixation, for certain parameter combinations a protected polymorphism and facultative fixation (either allele may become fixed according to initial frequencies) may both occur. Facultative fixation requires different selling rates for the dominant and recessive type. Protection of the polymorphism requires resource allocation for male and female function. In this connection the problem of purely genetically caused population extinction is discussed.
For general frequency dependence and regular segregation, the chances for establishment of a completely recessive gene are compared to those of a completely dominant gene. It is proven that the process of establishment of the recessive gene, despite a fitness advantage, may be considerably endangered by drift effects if random mating prevails. The recessive gene may reach the same effectivity in establishment as a dominant gene, only if the recessive homozygote mates exclusively with its own type during the period of establishment.