吉林省下盾螨属三新种（蜱螨亚纲：厉螨科）

吉林省已发现的下盾螨有溜下盾螨Ｈｙｐｏａｓｐｉｓ ｌｕｂｒｉｃａ Ｖｏｉｇｔｓ ｅｔ Ｏｕｄｅｍａｎｓ,力下盾螨Ｈｙｐｏａｓｐｉｓ ｈｒｄｙｉ Ｓａｍｓｉｎａｋ和尖狭下盾螨Ｈｙｐｏａｓｐｉｓ ａｃｕｌｅｉｆｅｒ（Ｃａｎｅｓｔｒｉｎｉ）。另外采到三新种,命名为松江下盾螨Ｈｙｐｏａｓｐｉｓ ｓｕｎｇａｒｉｓ ｓｐ．ｎｏｖ．,柔弱下盾螨Ｈｙｐｏａｓｐｉｓ ｄｅｂｉｌｉｓ ｓｐ．ｎｏｖ．和长毛下质螨Ｈｙｐ     

长岭下盾螨新种记录（蜱螨亚纲：厉螨科）

记述下盾螨属Ｈｙｐｏａｓｐｉｓ１新种，长岭下盾螨Ｈｙｐｏａｓｐｉｓ ｃｈａｎｇｌｉｎｇｅｎｓｉｓ，ｓｐ．ｎｏｖ．。模式标本存全疫布氏菌病防治基地（吉林省白城市）。     

吉林省下盾螨属一新种(蜱螨亚纲:革螨股)

记述下盾螨属一新种：白城下盾螨Ｈｙｐｏａｓｐｉｓ ｂａｉｃｈｅｎｇｅｎｓｉｓ Ｍａ,ｓｐ．ｎｏｖ．。     

湖南下盾螨属一新种记述(蜱螨亚纲:革螨股)

记述下盾螨属一新种,即湖南下盾螨Ｈｙｐｏａｓｐｉｓ ｈｕｎａｎｅｎｓｉｓ Ｍａ ｅｔ Ｚｈｅｎｇ,ｓｐ．ｎｏｖ．。     

下质螨属一新种（蜱螨亚纲：厉螨科）

本文记述ＱｕＪｉｎｇ下盾螨新种Ｈｙｐｏａｓｐｉｓ ｓｏｒｅｃｉｓ ｓｐ．ｎｏｖ．,并讨论与近似种Ｈ．ｈｒｙｄｉ Ｓａｍｓｉｎａｋ １９６１的鉴别特征。模式标本采自青海互助土族自治县的暗色ＱｕＪｉｎｇ Ｓｏｒｅｘ ｓｉｎａｌｉｓ Ｔｈｏｍａｓ,１９１２体上。文内量度单位均为μｍ,标本存放在青海省地方病防治研究所,西宁。     

派盾螨科二新种(蜱螨亚纲:革螨股)

本文记述派盾螨科２新种：副变革板螨Ｇａｍａｓｈｏｌａｓｐｉｓｐａｒａｖａｒｉａｂｉｌｉｓ,ｓｐ．ｎｏｖ．和陈氏讷派螨Ｎｅｐａｒｈｏｌａｓｐｉｓｃｈｅｎｐｅｎｇｉ,ｓｐ．ｎｏｖ．。模式标本存吉林省白城市全国鼠疫布氏菌病防治基地。     

维螨科一新种和巨螯螨科一新纪录（蜱螨亚纲：中气门亚目）

１９５９年４月在吉林省大安县安广，从黑线仓鼠巢采得维螨科一新种，命名为楔形维螨，新种Ｖｅｉｇａｉａ ｃｕｎｅａｔａ ｓｐ．ｎｏｖ．。同年９月在该县舍力从小家鼠巢采得巨螯螨科１中国新纪录，糙裂雕盾螨Ｇｌｙｐｔｈｏｌａｓｐｉｓ ａｓｐｅｒｒｉｍａ（Ｂｅｒｌｅｓｅ，１９０５）。     

维螨科—新种和巨螯螨科—新纪录──（蜱螨亚纲：中气门亚目）

１９５９年４月在吉林省大安县安广,从黑线仓鼠巢采得维螨科一新种,命名为楔形维螨,新种Ｖｅｉｇａｉａｃｕｎｅａｔａｓｐ．ｎｏｖ。同年９月在该县合力从小家鼠巢采得巨螯螨科１中国新纪录,糙裂雕盾螨Ｇｌｙｐｔｈｏｌａｓｐｉｓａｓｐｅｒｒｉｍａ（Ｂｅｒｌｅｓｅ,１９０５）。     

拟楔下盾螨后若螨和尖背广厉螨前若螨描述（蜱螨亚钢：厉螨科）

本文报道拟楔下盾螨ＨｙｐｏａｓｐｉｓｐａｒａｕｎｅｉｆｅｒＧｕｅｔＢａｉ后若螨和尖背广厉螨ＣｏｓｍｏｌａｅｌａｐｓａｃｕｔｉｓｃｕｔｕｓＴｅｎｇ前若螨,标本均采自吉林省白城的腐烂鼠巢。     

雕盾螨属一新种

本文记述雕盾螨属一新种,命名为白城雕盾螨Ｇｌｙｐｔｈｏｌａｓｐｉｓ ｂａｉｃｈｅｎｇｅｎｓｉｓ ｓｐ,ｎｏｖ采自腐烂鼠巢中。     

Quadruplexes of human telomere DNA analogs designed to contain G:A:G:A,G:G:A:A,and A:A:A:A tetrads

Replacement of two to four guanines by adenines in the human telomere DNA repeat dG₃(TTAG₃)₃ did not hinder the formation of quadruplexes if the substitutions took place in the terminal tetrad bridged by the diagonal loop of the intramolecular antiparallel three‐tetrad scaffold, as proved by CD and PAGE in both Na⁺ and K⁺ solutions. Thermodynamic data showed that, in Na⁺ solution, the dG₃(TTAG₃)₃ quadruplex was destabilized, the least by the two G:A:G:A tetrads, the most by the G:G:A:A tetrad in which the adenosines replaced syn‐guanosines. In physiological K⁺ solution, the highest destabilization was caused by the 4A tetrad. In K⁺, only the unmodified dG₃(TTAG₃)₃ quadruplex rearranged into a K⁺‐dependent quadruplex form, none of the multiple adenine‐modified structures did so. This may imply biological consequences for nonrepaired A‐for‐G mutations. © 2010 Wiley Periodicals, Inc. Biopolymers 93: 880–886, 2010.