海南省羊角蚱属二新种记述 (直翅目：蚱总科，刺翼蚱科)

 记述了海南省刺翼蚱科Scelimenidae羊角蚱属Criotettix两 新种：海南羊角蚱Criotettix hainanensis sp.nov.和长翅羊角蚱Criotettix longi pennis sp.nov.。模式标本保存在中山大学昆虫学研究所标本室。     

云南省西南部刺翼蚱科二新种(直翅目,蚱总科)

记述采自云南省西双版纳及普洱地区刺翼蚱科2新种,即三角羊角蚱 Criotettix triangularis sp.nov.,和长背赫蚱Hebarditettix dolichonota sp.nov.,模式标本保存于陕西师范大学动物研究所昆虫标本室.     

广西那坡地区蚱总科三新种(直翅目:蚱总科)

记述了刺翼蚱科及短翼蚱科昆虫3新种,即那坡羊角蚱Criotettix napoensis sp.nov.,黑胫波蚱Boli-varitettix nigritibialis sp.nov.及长翅大磨蚱Macromotettix longipennis sp.nov.。模式标本保存在中国科学院动物研究所。     

云南省西部地区蚱总科四新种记述(直翅目)

记述采自云南省西部地区蚱总科4新种,包括刺翼蚱科的盈江羊角蚱Criotettix yingjiangensis,sp.nov.和长背优角蚱Eucriotettix longidorsalis,sp.nov.,短翼蚱科的黑胫玛蚱Mazarredia nigritibia,sp.nov.及腾冲波蚱Bolivaritetti...     

广西刺翼蚱科新种记述(直翅目:蚱总科)

记述采自广西北海地区蚱总科昆虫2新种,即北海羊角蚱Criotettix beihaiensis sp.nov.,丘背羊角蚱Criotettix cliva sp.nov.,还记录了海南羊角蚱Criotettix hainanensis Liang雄性的新发现。模式标本保存在陕西师范大学动物研究所标本室。     

广西北部地区蚱总科七新种记述(直翅目)

记述采自广西北部蚱总科昆虫7新种,即黑股羊角蚱Criotettix nigrifemurus,sp．nov．,横刺羊角蚱Criotettix transpinius,sp．nov．,拟圆头波蚱Bolivaritettix circocephalosoides．sp．nov．,宜州蚱Tetrix yizhouensis,sp．nov．,丘背蚱Tetrix cliva,sp．nov．,白条悠背蚱Euparatettix albostriatus,sp．nov．及九万山悠背蚱Euparatettix jiuwanshanensis,sp．nov．。模式标本保存于陕西师范大学动物研究所昆虫标本室。     

海南省羊角蚱属二新种记述(直翅目：蚱总科，刺翼蚱科)

记述了海南省刺翼蚱科Scelimenidae羊角蚱属Criotettix两新种：海南羊角蚱Criotettix hainanensis sp.nov.和长翅羊角蚱Criotettix longi pennis sp.nov.。模式标本保存在中山大学昆虫学研究所标本室。     

云南省蚱总科四新种(直翅目)

记述采自云南省蚱总科昆虫4新种,即断脊羊角蚱Criotettix interrupticostus sp.nov.,勐腊波蚱Bolivaritettix menglaensis sp nov.,黄条尖顶蚱Teredorus flavistrial sp.nov.及昆明微翅蚱Alulatettix kunmingensissp.nov.,此外记述云南羊角蚱Criotettix yunnanensis Zheng et Ou及瘤背大磨蚱Macromotettix torulisinota Zheng的新发现的雄性.模式标本保存于陕西师范大学动物研究所昆虫标本室.     

云南省蚱总科两新种（直翅目）

记述采自云南省西双版纳傣族自治州及大理白族自治州蚱总科2新种,即瘤背羊角蚱Criotettix torulisinotus sp.nov,及保山蚱Tetrix baoshanensis sp.nov.,模式标本保存于陕西师范大学生命科学学院动物研究所标本室。     

广西羊角蚱属二新种记述(蚱总科:刺翼蚱科)

 记述采自广西壮族自治区羊角蚱属Criotettix Bolivar二新种,即断隆羊角蚱C.inte rrupta sp. Nov.及短翅羊角蚱C.brevipennis sp. Nov. 。模式标本保存于陕西师范 大学动物研究所标本室。     

Recent advances in the study of Kaposi's sarcoma-associated herpesvirus replication and pathogenesis

It has now been over twenty years since a novel herpesviral genome was identified in Kaposi's sarcoma biopsies. Since then, the cumulative research effort by molecular biologists, virologists, clinicians, and epidemiologists alike has led to the extensive characterization of this tumor virus, Kaposi's sarcoma-associated herpesvirus(KSHV; also known as human herpesvirus 8(HHV-8)), and its associated diseases. Here we review the current knowledge of KSHV biology and pathogenesis, with a particular emphasis on new and exciting advances in the field of epigenetics. We also discuss the development and practicality of various cell culture and animal model systems to study KSHV replication and pathogenesis.     

The structure, reproduction and taxonomy of Vidalia and Osmundaria (Rhodophyta, Rhodomelaceae)

Comprises species occurring mostly in subtidal habitats in tropical, subtropical and warm-temperate areas of the world. An analysis of the type species, V. spiralis (Sonder) Lamouroux ex J. Agardh, a species from Australia, establishes basic characters for distinguishing species in the genus. These characters are (1) branching patterns of thalli, (2) flat blades that may be spiralled on their axis, (3) width of the blade, (4) primary or secondary derivation of sterile and fertile branchlets and (5) position of sterile and fertile branchlets on the thalli. Application of the latter two characters provides an important basic method for separation of species into three major groups. Osmundaria , a genus known only in southern Australia, was studied in relation to Vidalia , and its separation from the Vidalia assemblage is not accepted. Species of Vidalia therefore are transferred to the older genus name, Osmundaria. Two new species, Osmundaria papenfussii and Osmundaria oliveae are described from Natal. Confusion in the usage of the epithet, Vidalia fimbriala Brown ex Turner has been clarified, and Vidalia gregaria Falkenberg, described as an epiphyte on Osmundaria pro/ifera Lamouroux, is revealed to be young branches of the host, Osmundaria prolifera.     

New or rare chromosome counts in Artemisia L. (Asteraceae, Anthemideae) and related genera from Kazakhstan

Fifteen chromosome counts of six Artemisia taxa and one species of each of the genera Brachanthemum, Hippolytia, Kaschgaria, Lepidolopsis and Turaniphytum are reported from Kazakhstan. Three of them are new reports, two are not consistent with previous counts and the remainder are confirmations of very scarce (one to four) earlier records. All the populations studied have the same basic chromosome number, x = 9, with ploidy levels ranging from 2x to 6x. Some correlations between ploidy level, morphological characters and distribution are noted.     

肝癌中HBV和HCV基因和抗原的分布及意义

采用原位分子杂交方法检测HCV RNA及HBV X基因;采用免疫组织化学方法研究HCV核心抗原,非结构区C33c抗原及HBxAg在肝细胞肝癌中的定位及分布.结果表明(1)HCV RNA、HBV X基因在肝细胞肝癌组织检出率分别为40%(55/136)和82%(112/136).HCV RNA定位于癌细胞的胞浆内,阳性细胞呈散在、灶状及弥漫分布三种形式;HBV X基因在肝癌细胞中的分布呈胞浆型、核型及核浆型,阳性细胞也呈上述三种分布形式;(2)HCV C33c抗原、核心抗原在肝细胞肝癌中的阳性率为81%(133/164)及86%(141/164).C33c抗原定位于癌细胞及肝细胞的胞浆内;核心抗原既定位于癌细胞核中,又可定位于胞浆中.C33c抗原阳性细胞以灶状分布为主;而核心抗原阳性细     

Selection with two alleles and complete dominance

For a plant selection model with frequency-independent viabilities, fertilities and selfing rates, it is shown that apart from global fixation, for certain parameter combinations a protected polymorphism and facultative fixation (either allele may become fixed according to initial frequencies) may both occur. Facultative fixation requires different selling rates for the dominant and recessive type. Protection of the polymorphism requires resource allocation for male and female function. In this connection the problem of purely genetically caused population extinction is discussed.
For general frequency dependence and regular segregation, the chances for establishment of a completely recessive gene are compared to those of a completely dominant gene. It is proven that the process of establishment of the recessive gene, despite a fitness advantage, may be considerably endangered by drift effects if random mating prevails. The recessive gene may reach the same effectivity in establishment as a dominant gene, only if the recessive homozygote mates exclusively with its own type during the period of establishment.