青海省纤恙螨属三新种（蜱螨亚纲：恙螨科）

本文描述采自青海的纤恙螨属Ｌｅｐｔｏｔｒｏｍｂｉｄｉｕｍ Ｎａｇａｙｏ ｅｔ ａｌ,１９１６,三个新种;巴颜纤恙螨Ｌ．（Ｌ．）ｂａｙａｎｅｎｓｉｓ ｓｐ．ｎｏｖ。,川纤恙螨Ｌ．（Ｌ．）ｈｕａｎｇｃｈｕａｎｅｎｓｉｓ ｓｐ．ｎｏｖ．,乌兰纤恙螨Ｌ．（Ｌ．）ｗｕｌａｎｅｎｓｉｓ ｓｐ．ｎｏｖ．模式标本均保存在青海省地方病防治研究所。     

春川恙螨属一新种：蜱螨亚纲：恙螨科

本文描述春川恙螨属Ｓｈｕｎｓｅｎｎｉａ Ｊａｍｅｓｏｎ ｅｔ Ｔｏｓｈｉｏｋａ一新种,即叉毛春川恙螨Ｓ．ｆｕｒｃｏｓｅｔａ ｓｐ．ｎｏｖ．,模式标本采自青海省化隆回簇自治区的大林姬鼠Ａｐｏｄｅｍｕｓ ｓｐｅｃｉｏｓｕｓ Ｔｅｍｍｉｎｃｋ,１８４５体长,文内量度单位均为μｍ。标本存放在青海省地方病防治研究所。     

春川恙螨属—新种(蜱螨亚纲:恙螨科)

本文描述春川恙螨属ＳｈｕｎｓｅｎｎｉａＪａｍｅｓｏｎｅｔＴｏｓｈｉｏｋａ一新种,即叉毛春川恙螨Ｓ．ｆｕｒｃｏｓｅｔａｓｐ．ｎｏｖ．,模式标本采自青海省化隆回族自治县的大林姬鼠ＡｐｏｄｅｍｕｓｓｐｅｃｉｏｓｕｓＴｅｍｍｉｎｃｋ,１８４５体长,文内量度单位均为μｍ。标本存放在青海省地方病防治研究所。     

纤恙螨属恙螨一新种：蜱螨亚纲：恙螨科

从采自新疆博乐县阿拉山口(海拔290 m)室内灰仓鼠伏龙芝亚种 Cricetulus migratorius caesius耳壳内的一批纤恙螨标本鉴定中发现中国新记录亚属——爱柯纤恙螨亚属 Ericotrombidium中的一个新种,定名为博乐纤恙螨 Leptotrombidium (E.) bolei sp.nov.,它与美丽纤恙螨L.(E.)pulchrum(Sosnina,1950)及索氏纤恙螨L.(E.)sokolovi Kudryshova,1984比较近似,但本新种IP及盾板量度除 AM外均较大,PW-SB≥PL,可与前者区别;IP较小,平均787,幅度763—821,DS较少,排列规则:2+8.6.6.4.2=28,可与后者区别。     

纤恙螨属一新种（蜱螨亚纲：恙螨科）

本文描述纤恙螨属,１９１６,一新种,五角纤恙螨,标本采自青海省玉树藏族自治州称多县。     

青海恙螨亚科三新种：蜱螨亚纲：恙螨科

本文记述了恙螨亚科Trombiculinae Ewing 1929的三个新种:1.球形钳齿恙螨,新种,Cheladonta globosea sp.nov..2.久治真棒恙螨,新种,Euschoengastia jiuzhiensis sp.nov.3.民和囊棒恙螨,新种Ascoschoengastia minheensis sp.nov.,分别采自青海省久治县、化隆县、民和县和互助县。     

无前恙螨属二新种(蜱螨目:恙螨科)

本描述了采自江苏黑线姬鼠体上的无前恙螨属二新种,即长盾无前恙螨Walchia(Walchia)longiscuta Xia et Chen,sp．nov．和孟氏无前恙螨Walchia(Walchia)meni Xiaet Chen,sp．nov．。模式标本保存于复旦大学上海医学院寄生虫学教研室。     

纤恙螨属一新种(蜱螨亚纲,恙螨科)

记述纤恙螨属Leptotrombidium Nagayo et al．,1916 1新种,即尖扎纤恙螨Leptotrombidium(L．) jianzhaensis sp．nov．,标本采自青海省黄南州尖扎县坎布拉地区红耳鼠兔Ochotona erythrotis体上。模式标本存放在青海省地方病预防控制所。文内量度单位均为μm。     

昆明西山纤恙螨一新种：真螨目：恙螨科

1958年在昆明西山采得1种恙螨,经鉴定系1新种,定名为昆西纤恙螨,标本保存在昆明军事医学研究所。昆西纤恙螨Leptotrombidlum(L.)kunxi Xiang et Wen,新种(图1-6) 标本:正模、幼虫,昆明西山,海拔2110m,1958-Ⅸ-26,向容炯、陈祖武采,宿主为普通树鼩中华亚种Tupaia glis chinenis,寄生在肛门周围。 鉴别:本新种与纤恙螨属的Leptotrombidium(L.)pesudofulmentum V.-G.et     

犹棒螨属一新种记述（蝉螨亚纲：恙螨科）

本文记述恙螨亚科ＴｒｏｍｂｉｃｕｌｉｎａｅＥｗｉｎｇ,犹棒螨属ＥｕｓｃｈｏｅｎｇａｓｔｉａＥｗｉｎｇ,祁连犹棒恙螨,新种Ｅｕｓｃｈｏｅｎｇａｓｔｉａｑｉｌｉａｎｅｎｓｉｓｓｐ．ｎｏｖ,采自青海省祁连县。模式标本保存在青海省地方病防治研究所。     

Recent advances in the study of Kaposi's sarcoma-associated herpesvirus replication and pathogenesis

It has now been over twenty years since a novel herpesviral genome was identified in Kaposi's sarcoma biopsies. Since then, the cumulative research effort by molecular biologists, virologists, clinicians, and epidemiologists alike has led to the extensive characterization of this tumor virus, Kaposi's sarcoma-associated herpesvirus(KSHV; also known as human herpesvirus 8(HHV-8)), and its associated diseases. Here we review the current knowledge of KSHV biology and pathogenesis, with a particular emphasis on new and exciting advances in the field of epigenetics. We also discuss the development and practicality of various cell culture and animal model systems to study KSHV replication and pathogenesis.     

The structure, reproduction and taxonomy of Vidalia and Osmundaria (Rhodophyta, Rhodomelaceae)

Comprises species occurring mostly in subtidal habitats in tropical, subtropical and warm-temperate areas of the world. An analysis of the type species, V. spiralis (Sonder) Lamouroux ex J. Agardh, a species from Australia, establishes basic characters for distinguishing species in the genus. These characters are (1) branching patterns of thalli, (2) flat blades that may be spiralled on their axis, (3) width of the blade, (4) primary or secondary derivation of sterile and fertile branchlets and (5) position of sterile and fertile branchlets on the thalli. Application of the latter two characters provides an important basic method for separation of species into three major groups. Osmundaria , a genus known only in southern Australia, was studied in relation to Vidalia , and its separation from the Vidalia assemblage is not accepted. Species of Vidalia therefore are transferred to the older genus name, Osmundaria. Two new species, Osmundaria papenfussii and Osmundaria oliveae are described from Natal. Confusion in the usage of the epithet, Vidalia fimbriala Brown ex Turner has been clarified, and Vidalia gregaria Falkenberg, described as an epiphyte on Osmundaria pro/ifera Lamouroux, is revealed to be young branches of the host, Osmundaria prolifera.     

New or rare chromosome counts in Artemisia L. (Asteraceae, Anthemideae) and related genera from Kazakhstan

Fifteen chromosome counts of six Artemisia taxa and one species of each of the genera Brachanthemum, Hippolytia, Kaschgaria, Lepidolopsis and Turaniphytum are reported from Kazakhstan. Three of them are new reports, two are not consistent with previous counts and the remainder are confirmations of very scarce (one to four) earlier records. All the populations studied have the same basic chromosome number, x = 9, with ploidy levels ranging from 2x to 6x. Some correlations between ploidy level, morphological characters and distribution are noted.     

肝癌中HBV和HCV基因和抗原的分布及意义

采用原位分子杂交方法检测HCV RNA及HBV X基因;采用免疫组织化学方法研究HCV核心抗原,非结构区C33c抗原及HBxAg在肝细胞肝癌中的定位及分布.结果表明(1)HCV RNA、HBV X基因在肝细胞肝癌组织检出率分别为40%(55/136)和82%(112/136).HCV RNA定位于癌细胞的胞浆内,阳性细胞呈散在、灶状及弥漫分布三种形式;HBV X基因在肝癌细胞中的分布呈胞浆型、核型及核浆型,阳性细胞也呈上述三种分布形式;(2)HCV C33c抗原、核心抗原在肝细胞肝癌中的阳性率为81%(133/164)及86%(141/164).C33c抗原定位于癌细胞及肝细胞的胞浆内;核心抗原既定位于癌细胞核中,又可定位于胞浆中.C33c抗原阳性细胞以灶状分布为主;而核心抗原阳性细     

Selection with two alleles and complete dominance

For a plant selection model with frequency-independent viabilities, fertilities and selfing rates, it is shown that apart from global fixation, for certain parameter combinations a protected polymorphism and facultative fixation (either allele may become fixed according to initial frequencies) may both occur. Facultative fixation requires different selling rates for the dominant and recessive type. Protection of the polymorphism requires resource allocation for male and female function. In this connection the problem of purely genetically caused population extinction is discussed.
For general frequency dependence and regular segregation, the chances for establishment of a completely recessive gene are compared to those of a completely dominant gene. It is proven that the process of establishment of the recessive gene, despite a fitness advantage, may be considerably endangered by drift effects if random mating prevails. The recessive gene may reach the same effectivity in establishment as a dominant gene, only if the recessive homozygote mates exclusively with its own type during the period of establishment.